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Nathan O. Stitziel

Researcher at Washington University in St. Louis

Publications -  101
Citations -  10114

Nathan O. Stitziel is an academic researcher from Washington University in St. Louis. The author has contributed to research in topics: Exome sequencing & Genome-wide association study. The author has an hindex of 35, co-authored 88 publications receiving 8282 citations. Previous affiliations of Nathan O. Stitziel include University of Illinois at Chicago & Harvard University.

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Loss-of-function mutations in APOC3, triglycerides, and coronary disease

Jacy R Crosby, +96 more
TL;DR: Rare mutations that disrupt AP OC3 function were associated with lower levels of plasma triglycerides and APOC3, and carriers of these mutations were found to have a reduced risk of coronary heart disease.
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A general approach to single-nucleotide polymorphism discovery

TL;DR: A unified approach to the discovery of variations in genetic sequence data of arbitrary DNA sources is presented, using the rapidly emerging genomic sequence as a template on which to layer often unmapped, fragmentary sequence data and to use base quality values to discern true allelic variations from sequencing errors.
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Exome sequencing identifies rare LDLR and APOA5 alleles conferring risk for myocardial infarction

Ron Do, +103 more
- 05 Feb 2015 - 
TL;DR: Kathiresan et al. as mentioned in this paper used exome sequencing of nearly 10,000 people to identify alleles associated with early-onset myocardial infarction; mutations in low-density lipoprotein receptor (LDLR) or apolipoprotein A-V (APOA5) were associated with disease risk.
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Exome-wide association study of plasma lipids in > 300,000 individuals

Dajiang J. Liu, +288 more
- 30 Oct 2017 - 
TL;DR: It is found that beta-thalassemia trait carriers displayed lower TC and were protected from coronary artery disease (CAD), and only some mechanisms of lowering LDL-C appeared to increase risk for type 2 diabetes (T2D); and TG-lowering alleles involved in hepatic production of TG-rich lipoproteins tracked with higher liver fat, higher risk for T2D, and lower risk for CAD.