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Nicolas Dupont

Researcher at University of Paris

Publications -  45
Citations -  7901

Nicolas Dupont is an academic researcher from University of Paris. The author has contributed to research in topics: Autophagy & Cilium. The author has an hindex of 18, co-authored 40 publications receiving 6214 citations. Previous affiliations of Nicolas Dupont include Paris Descartes University & Pasteur Institute.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
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Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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Galectin-3, a marker for vacuole lysis by invasive pathogens.

Irit Paz, +10 more
- 01 Apr 2010 - 
TL;DR: It is suggested that galectin‐3‐containing structures may serve as a potential novel tool to spot vacuole lysis following entry of Shigella into the cytosol and detected a striking phenomenon.
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ER–plasma membrane contact sites contribute to autophagosome biogenesis by regulation of local PI3P synthesis

Anna Chiara Nascimbeni, +10 more
- 14 Jul 2017 - 
TL;DR: It is demonstrated that E‐Syts are essential for autophagy‐associated PI3P synthesis at the cortical ER membrane via the recruitment of VMP1, the stabilizing ER partner of the PI3KC3 complex.
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Autophagy is required for endothelial cell alignment and atheroprotection under physiological blood flow

Anne-Clémence Vion, +36 more
- 10 Oct 2017 - 
TL;DR: It is demonstrated that inefficient autophagy contributes to the development of atherosclerotic plaques in low-SS areas, and the role of endothelial autophagic flux activation by SS as an atheroprotective mechanism is underline.