N
Ning Xia
Researcher at University of Mainz
Publications - 77
Citations - 5120
Ning Xia is an academic researcher from University of Mainz. The author has contributed to research in topics: Enos & Oxidative stress. The author has an hindex of 28, co-authored 70 publications receiving 3668 citations.
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Journal ArticleDOI
Roles of Vascular Oxidative Stress and Nitric Oxide in the Pathogenesis of Atherosclerosis
TL;DR: Prevention of vascular oxidative stress and improvement of endothelial NO production represent reasonable therapeutic strategies in addition to the treatment of established risk factors (hypercholesterolemia, hypertension, and diabetes mellitus).
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Antioxidant effects of resveratrol in the cardiovascular system
TL;DR: The antioxidant effects of resveratrol (3,5,4'‐trihydroxy‐trans‐stilbene) contribute substantially to the health benefits of this compound and are more likely to be attributable to its effect as a gene regulator.
Journal ArticleDOI
Cardiovascular effects and molecular targets of resveratrol.
TL;DR: In animal models of cardiovascular disease, resveratrol protects the heart from ischemia-reperfusion injury, reduces blood pressure and cardiac hypertrophy in hypertensive animals, and slows the progression of atherosclerosis.
Journal Article
Resveratrol reduces endothelial oxidative stress by modulating the gene expression of superoxide dismutase 1 (SOD1), glutathione peroxidase 1 (GPx1) and NADPH oxidase subunit (Nox4).
Gerrit Spanier,H Xu,Ning Xia,Silke Tobias,S Deng,Leszek Wojnowski,Ulrich Förstermann,Huige Li +7 more
TL;DR: The expressional suppression of pro-oxidative genes (such as NADPH oxidase) and induction of anti- oxidative enzymes ( such as SOD1 and GPx1) might be an important component of the vascular protective effect of resveratrol.
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Loneliness, Social Isolation, and Cardiovascular Health.
TL;DR: Repeated and chronic social stress leads to glucocorticoid resistance, enhanced myelopoiesis, upregulated proinflammatory gene expression, and oxidative stress, but the causal role of these mechanisms in the development of loneliness-associated CVD remains unclear.