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Pablo Iribarren

Researcher at National University of Cordoba

Publications -  40
Citations -  7502

Pablo Iribarren is an academic researcher from National University of Cordoba. The author has contributed to research in topics: Immune system & Inflammation. The author has an hindex of 18, co-authored 37 publications receiving 6089 citations. Previous affiliations of Pablo Iribarren include National Scientific and Technical Research Council.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Toll-like receptors in inflammation, infection and cancer.

TL;DR: The contribution of TLRs to diseases of the central nervous system (CNS), lung, gastrointestinal tract, kidney and skin as well as cancer is evaluated to provide new insight into the pathogenesis and progression of diseases and more importantly, into the potential for TLRs as targets of therapeutics.
Journal ArticleDOI

Autophagy in inflammation, infection, neurodegeneration and cancer.

TL;DR: The contribution of autophagy to inflammatory, infectious and neurodegenerative diseases, as well as cancer, is discussed.
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Toll-like receptors are key players in neurodegeneration

TL;DR: Results reported in the recent literature concerning the participation of Toll-like receptors in neurodegenerative diseases are discussed.