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Panagiota Mylona

Researcher at John Innes Centre

Publications -  5
Citations -  2983

Panagiota Mylona is an academic researcher from John Innes Centre. The author has contributed to research in topics: Root hair & Gene. The author has an hindex of 5, co-authored 5 publications receiving 2810 citations. Previous affiliations of Panagiota Mylona include Norwich Research Park & Sainsbury Laboratory.

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Reactive oxygen species produced by nadph oxidase regulate plant cell growth

TL;DR: It is shown here that RHD2 is an NADPH oxidase, a protein that transfers electrons from NADPH to an electron acceptor leading to the formation of reactive oxygen species (ROS) and that ROS accumulate in growing wild-type (WT) root hairs but their levels are markedly decreased in rhd2 mutants.
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Symbiotic Nitrogen Fixation.

TL;DR: This review describes the early steps of the interaction between rhizobia and legumes that result in the formation of a nitrogen-fixing nodule and focuses on the role of specific lipooligosaccharides secreted by Rhizobia in the induction of these early steps.
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Sad3 and Sad4 Are Required for Saponin Biosynthesis and Root Development in Oat

TL;DR: This study focuses on mutations at the other two loci, Sad3 and Sad4, which result in stunted root growth, membrane trafficking defects in the root epidermis, and root hair deficiency, and various lines of evidence indicate that these effects are dosage-dependent.
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SCHIZORIZA controls an asymmetric cell division and restricts epidermal identity in the Arabidopsis root.

TL;DR: Schizoriza (scz), a mutant with defective radial patterning, has two distinct roles: (1) it acts as a suppressor of epidermal fate in the ground tissue, and (2) it is required to repress periclinal divisions in the meristem.
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High throughput screening of mutants of oat that are defective in triterpene synthesis

TL;DR: High throughput screening is carried out to identify mutants of diploid oat (Avena strigosa) that are blocked in the early steps of triterpene synthesis and shows that mutants that are affected in the first committed step in synthesis of beta-amyrin-derived triterpenes, and so are unable to cyclise 2,3-oxidosqualene to beta- amyrin (sad1 mutants), accumulate elevated levels of primary sterols.