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Paolo Tucci

Researcher at University of Foggia

Publications -  69
Citations -  1454

Paolo Tucci is an academic researcher from University of Foggia. The author has contributed to research in topics: Internal medicine & Medicine. The author has an hindex of 21, co-authored 56 publications receiving 1120 citations. Previous affiliations of Paolo Tucci include Sapienza University of Rome.

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Soluble βamyloid1-42: a critical player in producing behavioural and biochemical changes evoking depressive-related state?

TL;DR: The effects of soluble Aβ1‐42 on working memory and depressive/anxiety‐related behaviour in rats and on 5‐hydroxytryptaminergic neurotransmission and neurotrophin content in various brain regions are investigated.
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The NADPH Oxidase NOX2 Controls Glutamate Release: A Novel Mechanism Involved in Psychosis-Like Ketamine Responses

TL;DR: The results suggest that NOX2 is a major source of ROS production in the prefrontal cortex controlling glutamate release and associated behavioral alterations after acute ketamine exposure, and may lead to neuroadaptative downregulation of NMDA receptor subunits.
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Carvacrol: From Ancient Flavoring to Neuromodulatory Agent

TL;DR: The data suggest that carvacrol is a brain-active molecule that clearly influences neuronal activity through modulation of neurotransmitters and might determine feelings of well-being and could possibly have positive reinforcer effects.
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Neuroendocrine profile in a rat model of psychosocial stress: relation to oxidative stress.

TL;DR: It is demonstrated that psychosocial stress induces early elevation of NOX2-derived oxidative stress in the hypothalamus and consequent alterations of the HPA-axis, leading ultimately to an altered behavior.
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Clastogenicity of atrazine assessed with the Allium cepa test

TL;DR: The present study was undertaken to add new data on the genotoxic potential of atrazine using the Allium cepa chromosome aberration test, which detected the clastogenicity ofatrazine at concentrations that are likely to be encountered in water, a common site of atazine contamination.