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Pedro R. Moreno

Researcher at Mount Sinai Hospital

Publications -  87
Citations -  6876

Pedro R. Moreno is an academic researcher from Mount Sinai Hospital. The author has contributed to research in topics: Percutaneous coronary intervention & Conventional PCI. The author has an hindex of 27, co-authored 78 publications receiving 6264 citations. Previous affiliations of Pedro R. Moreno include University of Kentucky & Boston Medical Center.

Papers
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Journal ArticleDOI

From vulnerable plaque to vulnerable patient: a call for new definitions and risk assessment strategies: Part II.

Morteza Naghavi, +63 more
- 07 Oct 2003 - 
TL;DR: The term "vulnerable patient" may be more appropriate and is proposed now for the identification of subjects with high likelihood of developing cardiac events in the near future and a quantitative method for cumulative risk assessment of vulnerable patients needs to be developed.
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Terminology for high-risk and vulnerable coronary artery plaques

TL;DR: A group of investigators met for two days in Santorini, Greece, to discuss progress in the field of identification and treatment of high risk/vulnerable atherosclerotic plaques and patients and reached agreement on their own usage of the terms as described below.
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Coronary Composition and Macrophage Infiltration in Atherectomy Specimens From Patients With Diabetes Mellitus

TL;DR: Coronary tissue from patients with diabetes exhibits a larger content of lipid-rich atheroma, macrophage infiltration, and subsequent thrombosis than tissue from Patients without diabetes, suggesting an increased vulnerability for coronary thromBosis in patients with Diabetes mellitus.
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Macrophages, smooth muscle cells, and tissue factor in unstable angina : Implications for cell-mediated thrombogenicity in acute coronary syndromes

TL;DR: Tissue factor content is increased in unstable angina and correlates with areas of macrophages and smooth muscle cells, suggesting a cell-mediated thrombogenicity in patients with acute coronary syndromes.
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New aspects in the pathogenesis of diabetic atherothrombosis.

TL;DR: Three subfamilies, including PPAR-alpha, -delta, and -gamma, have been identified with crucial roles in lipid metabolism, plaque inflammation, expression of adhesion molecules and cytokines, and regulation of matrix metalloproteinases.