P
Peter Möller
Researcher at Heidelberg University
Publications - 136
Citations - 8547
Peter Möller is an academic researcher from Heidelberg University. The author has contributed to research in topics: Antigen & B cell. The author has an hindex of 42, co-authored 136 publications receiving 8424 citations. Previous affiliations of Peter Möller include University of Würzburg & University of Ulm.
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Journal ArticleDOI
Monoclonal antibody-mediated tumor regression by induction of apoptosis
B. C. Trauth,C. Klas,Anke M.J. Peters,Siegfried Matzku,Peter Möller,Werner Falk,Klaus-Michael Debatin,Peter H. Krammer +7 more
TL;DR: Histological thin sections of the regressing tumor showed that anti-APO-1 was able to induce apoptosis in vivo, suggesting induction of apoptosis as a consequence of a signal mediated through cell surface molecules like APO- 1 may be a useful therapeutic approach in treatment of malignancy.
Journal Article
Constitutive and induced expression of APO-1, a new member of the nerve growth factor/tumor necrosis factor receptor superfamily, in normal and neoplastic cells.
Frank Leithäuser,Jens Dr Dhein,Gunhild Mechtersheimer,Karin Koretz,S. Brüderlein,C. Henne,Annette D. Schmidt,Klaus-Michael Debatin,Peter H. Krammer,Peter Möller +9 more
TL;DR: Tissue distribution, in vitro expression, and reaction upon cytokine-induced activation suggest that APO-1 might not only transmit apoptotic signals but might play a more general role in growth control.
Journal ArticleDOI
T Cell Awareness of Paternal Alloantigens During Pregnancy
TL;DR: During pregnancy a semiallogeneic fetus survives despite the presence of maternal T cells specific for paternally inherited histocompatibility antigens, and during pregnancy maternal T Cells acquire a transient state of tolerance specific for paternal alloantigens.
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Estimation of surface area and length with the orientator.
TL;DR: The orientator is an unbiased, design‐based approach to the quantitative study of anisotropic structures such as muscle, myocardium, bone and cartilage, and generation of isotropic sections for second‐order stereology (three‐dimensional pattern analysis).
Journal ArticleDOI
Expression of APO-1 (CD95), a member of the NGF/TNF receptor superfamily, in normal and neoplastic colon epithelium.
Peter Möller,Karin Koretz,Frank Leithäuser,S. Brüderlein,C. Henne,A. Quentmeier,Peter H. Krammer +6 more
TL;DR: It is concluded that neoplastic transformation of colon epithelium often leads to a loss of the physiologic, high level of surface APO‐I by giving rise either to a stable lack of APo‐I or to an IFN‐γ/TNF‐α‐sensitive phenotype of inducible APO'I expression.