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Robert Fagard

Researcher at Katholieke Universiteit Leuven

Publications -  788
Citations -  109235

Robert Fagard is an academic researcher from Katholieke Universiteit Leuven. The author has contributed to research in topics: Blood pressure & Ambulatory blood pressure. The author has an hindex of 114, co-authored 787 publications receiving 104613 citations.

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Relationships of heart rate and heart rate variability with conventional and ambulatory blood pressure in the population.

TL;DR: Relationships between autonomic nervous function at rest, assessed by use of power spectral analysis of heart rate variability, and conventional blood pressure, can at least partly be ascribed to the influence of the measurement conditions, whereas the orthostatic autonomic responses appear to be influenced by blood pressure per se.
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Host and environmental determinants of polychlorinated aromatic hydrocarbons in serum of adolescents.

TL;DR: It was showed that in 16- to 18-year-old teenagers host factors are important determinants of serum concentrations of PCAHs, whereas environmentally related determinants may to some extent contribute independently to human exposure to these persistent chemicals in the environment.
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Familial aggregation of blood pressure, anthropometric characteristics and urinary excretion of sodium and potassium—A population study in two Belgian towns

TL;DR: The tendency of the correlation coefficients for body weight and body mass index to be higher in mother-offspring than in father-offpring pairs suggests a closer maternal role in the determination of calorie intake in offspring.
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Plasma renin system during exercise in normal men

TL;DR: At rest as well as during exercise, Aldo rose with increasing ANG II, but the stimulatory effect of ANG II on Aldo was attenuated with higher sodium intake, as estimated from UVNa, independent of the level of physical activity.
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Acute and chronic systemic and pulmonary hemodynamic effects of angiotensin converting enzyme inhibition with captopril in hypertensive patients

TL;DR: The increase in cardiac output during long-term treatment seems to be associated with a hypermetabolic state and in patients with very severe hypertension, with restoration to normal of mildly decreased left ventricular function.