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Robert Tibshirani

Researcher at Stanford University

Publications -  620
Citations -  359457

Robert Tibshirani is an academic researcher from Stanford University. The author has contributed to research in topics: Lasso (statistics) & Gene expression profiling. The author has an hindex of 147, co-authored 593 publications receiving 326580 citations. Previous affiliations of Robert Tibshirani include University of Toronto & University of California.

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Recent results of elective open cholecystectomy in a North American and a European center. Comparison of complications and risk factors.

TL;DR: Elective open cholecystectomy is a safe procedure, particularly in terms of highly morbid complications and death, and the data demonstrate the advantages of a uniform way of reporting surgical complications, which may permit meaningful comparisons among centers.
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"Preconditioning" for feature selection and regression in high-dimensional problems

TL;DR: This work proposes a method for variable selection that first estimates the regression function, yielding a "preconditioned" response variable, and shows that under a certain Gaussian latent variable model, application of the LASSO to the preconditioned response variable is consistent as the number of predictors and observations increases.
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Boolean implication networks derived from large scale, whole genome microarray datasets

TL;DR: A meta-analysis of data from thousands of microarrays for humans, mice, and fruit flies finds millions of implication relationships between genes that would be missed by other methods.
Proceedings Article

Discriminant Adaptive Nearest Neighbor Classification and Regression

TL;DR: A locally adaptive form of nearest neighbor classification is proposed to try to finesse this curse of dimensionality, and a method for global dimension reduction, that combines local dimension information is proposed.
Journal ArticleDOI

Alteration of the lipid profile in lymphomas induced by MYC overexpression

TL;DR: A relationship between the appearance of specific lipid species and the overexpression of MYC in lymphomas is suggested, including many of the lipid species identified as significant for MYC-induced animal lymphoma tissue.