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Robin M. Murray

Researcher at King's College London

Publications -  1583
Citations -  128883

Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.

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Preterm birth and structural brain alterations in early adulthood

TL;DR: The results reveal that VPT birth continues to be associated with altered structural brain anatomy in early adult life, although it remains to be ascertained whether these changes reflect neurodevelopmental delays or long lasting structural alterations due to prematurity.
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Women with schizophrenia: pregnancy outcome and infant death among their offspring.

TL;DR: It is concluded that schizophrenia in the mother implies an increased risk for poor perinatal outcome, not fully explained by maternal factors, and a need to consider a common familial (probably genetic) vulnerability for pre- and per inatal stress and schizophrenia.
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Gender Differences in Incidence and Age at Onset of Mania and Bipolar Disorder Over a 35-Year Period in Camberwell, England

TL;DR: Men appear to have an earlier onset of mania and bipolar disorder than women, and the association of male gender and childhood antisocial behavior with early-onset bipolar disorder raised the possibility of the existence of an early-ONSet subgroup.
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Identification of a novel neuregulin 1 at-risk haplotype in Han schizophrenia Chinese patients, but no association with the Icelandic/Scottish risk haplotype.

TL;DR: Different haplotypes within the boundaries of the NRG1 gene may be associated with schizophrenia in the Han Chinese, including markers of a seven-marker haplotype at the 5′end of the gene found to be in excess in Icelandic and Scottish schizophrenia patients.
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Schizophrenia: from developmental deviance to dopamine dysregulation.

TL;DR: Two major theories of schizophrenia are respectively, the neurodevelopmental hypothesis and the dopamine hypothesis, which suggest that excess striatal dopamine is responsible for increased salience being given to insignificant events and thoughts and that this underpins the development of psychotic symptoms.