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Robin M. Murray

Researcher at King's College London

Publications -  1583
Citations -  128883

Robin M. Murray is an academic researcher from King's College London. The author has contributed to research in topics: Psychosis & Schizophrenia. The author has an hindex of 171, co-authored 1539 publications receiving 116362 citations. Previous affiliations of Robin M. Murray include University of Cambridge & National Institutes of Health.

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Cardiovascular risk factors and metabolic syndrome in people with established psychotic illnesses

TL;DR: In this paper, a large randomized controlled trial (IMPaCT RCT) was conducted to determine the prevalence of cardiometabolic risk factors and establish the proportion of people with psychosis meeting criteria for the metabolic syndrome (MetS).
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Insight, grey matter and cognitive function in first-onset psychosis.

TL;DR: Reduced general neuropsychological function was linked to poor symptom relabelling ability and the cingulate gyrus (as part of a midline cortical system) along with right hemisphere regions may be involved in illness and symptom self-appraisal in first-onset psychosis.
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Effect of symptoms on executive function in bipolar illness

TL;DR: Executive function deficits in bipolar I disorder are most evident during mania, and are particularly associated with formal thought disorder, however, deficits in response initiation, strategic thinking and inhibitory control may be more related to the underlying disorder than a particular symptom profile.
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Antisaccade performance in monozygotic twins discordant for schizophrenia: the Maudsley twin study.

TL;DR: Antisaccade spatial accuracy and latency deficits may serve as markers of genetic liability for schizophrenia and be correlated with negative symptoms in the patients.
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Lack of normal pattern of cerebral asymmetry in familial schizophrenic patients and their relatives--The Maudsley Family Study.

TL;DR: The findings indicate that lack of the normal pattern of frontal and occipital asymmetry is a marker for genetic liability to schizophrenia in families multiply affected with schizophrenia.