R
Roger J. Keynes
Researcher at University of Cambridge
Publications - 110
Citations - 9316
Roger J. Keynes is an academic researcher from University of Cambridge. The author has contributed to research in topics: Somite & Growth cone. The author has an hindex of 48, co-authored 109 publications receiving 9110 citations. Previous affiliations of Roger J. Keynes include Laboratory of Molecular Biology & Guy's Hospital.
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Peripheral nerve regeneration
James W. Fawcett,Roger J. Keynes +1 more
TL;DR: Basal lamina grafts for reconnecting severed nerves are prepared from muscle by removing cellular material therefrom while preserving the tubular structure of the basal lamina, eventually reestablishing nerve function through the regenerated graft.
Journal ArticleDOI
Segmental patterns of neuronal development in the chick hindbrain.
Andrew Lumsden,Roger J. Keynes +1 more
TL;DR: Identification of specific neuronal populations and their projections in the developing hindbrain reveals a segmental organization in which pairs of metameric epithelial units cooperate to generate the repeating sequence of cranial branchiomotor nerves.
Journal ArticleDOI
Segmentation in the chick embryo hindbrain is defined by cell lineage restrictions
TL;DR: It is shown here that the rhombomere boundaries are partitions across which cells do not move, raising the possibility that they are analogous to the compart-ments of insects.
Journal ArticleDOI
Segmentation in the vertebrate nervous system
Roger J. Keynes,Claudio D. Stern +1 more
TL;DR: Zinc iodideosmium tetroxide staining of chick embryos reveals that motor and sensory axons grow from the neural tube region through the anterior (rostral) half of each successive somite, showing that neural segmentation is not intrinsic to the Neural tube.
Journal ArticleDOI
The migration of neural crest cells and the growth of motor axons through the rostral half of the chick somite.
TL;DR: It is found that the extracellular matrix components fibronectin and laminin are evenly distributed between the rostral and caudal halves of the somite, and neither of these molecules plays a critical role in determining the specific pathway of neural crest cells or motor axons through the roStral half of the Somite.