Ronald Williams

TL;DR: Air pollution monitoring paradigm is rapidly changing due to recent advances in the development of portable, lower-cost air pollution sensors reporting data in near-real time at a high-time resolution, increased computational and visualization capabilities, and wireless communication/infrastructure.

TL;DR: The observations in young, healthy, nonsmoking, male North Carolina Highway Patrol troopers suggest that in-vehicle exposure to PM(2.5) may cause pathophysiologic changes that involve inflammation, coagulation, and cardiac rhythm.

TL;DR: Over the past decade, a range of sensor technologies became available on the market, enabling a revolutionary shift in air pollution monitoring and assessment, and it can be argued that with a significant future expansion of monitoring networks, including indoor environments, there may be less need for wearable or portable sensors/monitors to assess personal exposure.

TL;DR: A spline mixed-effects model revealed a negative relationship between outdoor 24-h average fine particulate matter and high-frequency HRV that was consistent with an earlier Baltimore study and is consistent with earlier findings that exposures to PM2.5 are associated with decreased HRV in the elderly.

TL;DR: PM2.5 originating from speed-changing traffic modulates the autonomic control of the heart rhythm, increases the frequency of premature supraventricular beats and elicits pro-inflammatory and pro-thrombotic responses in healthy young men.