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Salvatore G. Caradonna

Researcher at Rockefeller University

Publications -  5
Citations -  22

Salvatore G. Caradonna is an academic researcher from Rockefeller University. The author has contributed to research in topics: Biology & Epigenetics. The author has co-authored 1 publications.

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Genomic modules and intramodular network concordance in susceptible and resilient male mice across models of stress.

TL;DR: In this article, the authors investigated the genomic signatures in the ventral hippocampus common to mouse models of stress using RNA-sequencing and found that differentially expressed genes (DEGs) were concordant for gene networks involved in neurotransmission, cytoskeleton function, and vascularization.
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Corticosterone induces discrete epigenetic signatures in the dorsal and ventral hippocampus that depend upon sex and genotype: focus on methylated Nr3c1 gene

TL;DR: In this article , the authors investigated the genomic signatures of oral corticosterone in the dHPC and vHPC of WT and hMet male and female mice, and examined sex and genotype differences in response to oral CORT.
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An allostatic epigenetic memory on chromatin footprints after double-hit acute stress

TL;DR: This paper investigated the behavioral traits and epigenetic signatures in a double-hit mouse model of acute stress in which heterotypic stressors (acute swim stress and acute restraint stress) were applied within a 7-day interval period.
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Got Milk? Maternal immune activation during the mid-lactational period affects nutritional milk quality and adolescent offspring sensory processing in male and female rats

TL;DR: This article investigated how lipopolysaccharide (LPS)-induced maternal immune activation (MIA) on postnatal day (P)10 affects maternal care, milk, and offspring development and found that MIA was associated with elevated milk corticosterone concentrations on P10, which recovered by P11.
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P686. Chromatin Footprints in the Ventral Hippocampus in a Double-Hit Model of Heterotypic Stress

TL;DR: In this article , the epigenetic mechanisms that underlie persistent genome-wide signatures of stress coping were examined, showing that a single exposure to acute stress has long-term functional and structural consequences in the hippocampus.