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Santanu Raychaudhuri

Researcher at University of California, Los Angeles

Publications -  16
Citations -  949

Santanu Raychaudhuri is an academic researcher from University of California, Los Angeles. The author has contributed to research in topics: Viral replication & Internal ribosome entry site. The author has an hindex of 13, co-authored 16 publications receiving 846 citations.

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Histone Code Modifications Repress Glucose Transporter 4 Expression in the Intrauterine Growth-restricted Offspring

TL;DR: It is concluded that epigenetic mechanisms consisting of histone code modifications repress skeletal muscle glut4 transcription in the postnatal period and persist in the adult female IUGR offspring.
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The heat shock protein inhibitor Quercetin attenuates hepatitis C virus production.

TL;DR: The marked inhibition of virus production by Quercetin may partially be related to reduction of HSP40 and HSP70 and their potential involvement in IRES translation, as well as viral morphogenesis or secretion.
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Glucose transporter isoform-3 mutations cause early pregnancy loss and fetal growth restriction.

TL;DR: It is concluded that GLUT3 mutations cause a gene dose-dependent early pregnancy loss or late-gestation fetal growth restriction despite the presence of embryonic and placental GLUT1 and a compensatory increase in system A amino acid placental transport.
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Nuclear entry of poliovirus protease-polymerase precursor 3CD: implications for host cell transcription shut-off.

TL;DR: It is demonstrated that both 3CD and 3D are capable of entering the nucleus in PV-infected cells, however, both polypeptides remain in the cytoplasm in uninfected HeLa cells, suggesting that both the 3D NLS and PV infection are required for the entry of 3CD into the nucleus.
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Divergent antiviral effects of bioflavonoids on the hepatitis C virus life cycle.

TL;DR: It is demonstrated that catechin, naringenin, and quercetin possess significant antiviral activity, with no associated cytotoxicity, and combination of these bioflavonoids may act synergistically against HCV.