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Simone Appenzeller

Researcher at State University of Campinas

Publications -  324
Citations -  5850

Simone Appenzeller is an academic researcher from State University of Campinas. The author has contributed to research in topics: Medicine & Internal medicine. The author has an hindex of 38, co-authored 266 publications receiving 4644 citations. Previous affiliations of Simone Appenzeller include McGill University Health Centre & McGill University.

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Neurologic involvement in scleroderma: a systematic review.

TL;DR: Previously considered a rare event, nervous system involvement in scleroderma has been increasingly recognized, and seizures and headache are the most reported features in LS en coup de sabre, while peripheral and autonomic nervous systems involvement predominate in SSc.
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An open, multi-vendor, multi-field-strength brain MR dataset and analysis of publicly available skull stripping methods agreement.

TL;DR: An open, multi‐vendor, multi-field strength magnetic resonance (MR) T1‐weighted volumetric brain imaging dataset, named Calgary‐Campinas‐359 (CC‐359), indicated that vendor and magnetic field strength have statistically significant impacts on skull stripping results.
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Epileptic seizures in systemic lupus erythematosus

TL;DR: Epileptic seizures were observed in 11.2% of systemic lupus erythematosus (SLE) patients, and patients with renal flares, epileptic seizures at SLE disease onset, and antiphospholipid antibodies were at greater risk for acute symptomatic seizures during follow-up.
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Seizure frequency and lateralization affect progression of atrophy in temporal lobe epilepsy.

TL;DR: The progression of white and gray matter atrophy in patients with mesial temporal lobe epilepsy (MTLE) was more intense in Patients with left MTLE and was associated with poorer seizure control and a longer duration of epilepsy.
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Risk factors in cardiovascular disease in systemic lupus erythematosus.

TL;DR: Empirical observations suggest that, together with classical conventional risk factors, other mechanisms promote accelerated atherosclerosis in inflammatory diseases like SLE.