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Subhabrata Sanyal
Researcher at Biogen Idec
Publications - 46
Citations - 2000
Subhabrata Sanyal is an academic researcher from Biogen Idec. The author has contributed to research in topics: Drosophila Protein & Genetic model. The author has an hindex of 24, co-authored 46 publications receiving 1783 citations. Previous affiliations of Subhabrata Sanyal include University of Arizona & Emory University.
Papers
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Journal ArticleDOI
Plasticity of local GABAergic interneurons drives olfactory habituation
Sudeshna Das,Madhumala K. Sadanandappa,Adrian Dervan,Aoife Larkin,John Anthony Lee,Indulekha P. Sudhakaran,Rashi Priya,Raheleh Heidari,Eimear E. Holohan,Angel Pimentel,Avni V. Gandhi,Kei Ito,Subhabrata Sanyal,Jing W. Wang,Veronica Rodrigues,Mani Ramaswami +15 more
TL;DR: Evidence that habituation arises from potentiation of inhibitory transmission within a circuit motif commonly repeated in the nervous system is presented and it is suggested that similar circuit mechanisms may operate in other species and sensory systems.
Journal ArticleDOI
AP-1 functions upstream of CREB to control synaptic plasticity in Drosophila
TL;DR: In this paper, the authors analyse cellular functions and regulation of the best known immediate-early transcription factor, AP-1; a heterodimer of the basic leucine zipper proteins Fos and Jun.
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The translational repressor Pumilio regulates presynaptic morphology and controls postsynaptic accumulation of translation factor eIF-4E.
Kaushiki P. Menon,Subhabrata Sanyal,Yasuaki Habara,Ricardo Sanchez,Robin P. Wharton,Mani Ramaswami,Kai Zinn +6 more
TL;DR: It is shown that Pum is an important mediator of synaptic growth and plasticity at the neuromuscular junction (NMJ), and results suggest that postsynaptic Pum modulates synaptic function via direct control of eIF-4E expression.
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Genomic mapping and expression patterns of C380, OK6 and D42 enhancer trap lines in the larval nervous system of Drosophila.
TL;DR: The mapping of three GAL4 lines commonly used as motor neuron drivers are reported, suggesting conserved control of Futsch expression across species, most likely through upstream cis-acting elements.
Journal ArticleDOI
Sleep Fragmentation and Motor Restlessness in a Drosophila Model of Restless Legs Syndrome
Amanda A. H. Freeman,Elaine L. Pranski,R. Daniel Miller,Sara Radmard,Douglas Bernhard,Hyder A. Jinnah,Ranjita Betarbet,David B. Rye,Subhabrata Sanyal +8 more
TL;DR: It is reported that loss of the Drosophila homolog CG1826 (dBTBD9) appreciably disrupts sleep with concomitant increases in waking and motor activity and it is shown that BTBD9 regulates brain dopamine levels in flies and controls iron homeostasis through the iron regulatory protein-2 in human cell lines.