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Susan M. Rivera

Researcher at University of California, Davis

Publications -  122
Citations -  5611

Susan M. Rivera is an academic researcher from University of California, Davis. The author has contributed to research in topics: Fragile X syndrome & FMR1. The author has an hindex of 37, co-authored 112 publications receiving 5084 citations. Previous affiliations of Susan M. Rivera include Stanford University & University of California, Berkeley.

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Developmental Changes in Mental Arithmetic: Evidence for Increased Functional Specialization in the Left Inferior Parietal Cortex

TL;DR: Evidence is provided for a process of increased functional specialization of the left inferior parietal cortex in mental arithmetic, a process that is accompanied by decreased dependence on memory and attentional resources with development.
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Dissociating Prefrontal and Parietal Cortex Activation during Arithmetic Processing

TL;DR: Functional magnetic resonance imaging findings indicate a dissociation in prefrontal and parietal cortex function during arithmetic processing and provide the first evidence for a specific role for the angular gyrus in arithmetic computation independent of other processing demands.
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Cortical Folding Abnormalities in Autism Revealed by Surface-Based Morphometry

TL;DR: Findings are consistent with evidence of an altered trajectory of early brain development in autism, and they identify several regions that may have abnormal patterns of connectivity in individuals with autism.
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A Randomized Double-Blind, Placebo-Controlled Trial of Minocycline in Children and Adolescents with Fragile X Syndrome

TL;DR: Minocycline treatment for 3 months in children with FXS resulted in greater global improvement than placebo, and treatment for3 months appears safe; however, longer trials are indicated to further assess benefits, side effects, and factors associated with a clinical response to minocyCline.
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A functional and structural study of emotion and face processing in children with autism.

TL;DR: The results showed that children with autism exhibited intact emotion matching, while showing diminished activation of the fusiform gyrus (FG) and the amygdala, consistent with abnormalities in circuits involved in emotion and face processing reported in studies of older subjects with autism.