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Suzanne Oparil

Researcher at University of Alabama at Birmingham

Publications -  941
Citations -  122414

Suzanne Oparil is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Blood pressure & Angiotensin II. The author has an hindex of 106, co-authored 885 publications receiving 113983 citations. Previous affiliations of Suzanne Oparil include Michigan State University & Oregon Health & Science University.

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Journal ArticleDOI

Weight Reduction for Obesity-Induced Heart Failure with Preserved Ejection Fraction.

Karnika Ayinapudi, +4 more
- 03 Jul 2020 - 
TL;DR: Both substantial weight Reduction with bariatric surgery and lesser levels of weight reduction with caloric restriction are promising therapeutic approaches to obesity-induced heart failure with preserved ejection fraction.
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Spironolactone Reduces Aortic Stiffness in Patients With Resistant Hypertension Independent of Blood Pressure Change.

Sudeep Aryal, +14 more
- 07 Sep 2021 - 
TL;DR: Aortic stiffness is an independent predictor of cardiovascular events in patients with arterial hypertension as mentioned in this paper, which is often linked to hyperaldosteronism and associated hyperthyroidism.
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Distinctive Risk Factors and Phenotype of Younger Patients With Resistant Hypertension: Age Is Relevant.

Lama Ghazi, +4 more
- 01 Jan 2017 - 
TL;DR: In this paper, the authors conducted a cross-sectional analysis with predefined age groups of a large, ethnically diverse cohort of 2170 patients referred to the Hypertension Clinic at the University of Alabama at Birmingham.
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H29: Comparison of the efficacy of two angiotensin II antagonists, valsartan and losartan, in essential hypertension

Pascale Oddou-Stock, +7 more
- 01 Apr 1997 - 
Journal ArticleDOI

Captopril and the response to stress in the spontaneously hypertensive rat.

Kathleen H. Berecek, +5 more
- 01 Feb 1988 - 
TL;DR: The results suggest that chronic intracerebroventricular administration of captopril, through blockade of the brain renin-angiotensin system, alters the hormonal response of SHR to stress.