S
Suzanne Oparil
Researcher at University of Alabama at Birmingham
Publications - 941
Citations - 122414
Suzanne Oparil is an academic researcher from University of Alabama at Birmingham. The author has contributed to research in topics: Blood pressure & Angiotensin II. The author has an hindex of 106, co-authored 885 publications receiving 113983 citations. Previous affiliations of Suzanne Oparil include Michigan State University & Oregon Health & Science University.
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Nitric oxide mediates immune dysfunction in the spontaneously hypertensive rat.
TL;DR: Evidence is provided that the immune depression observed in the spontaneously hypertensive rat is nitric oxide dependent, and it is demonstrated that L-arginine is required for the development of the depressed mitogen-induced proliferative responses in these cells.
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2013 European Society of Hypertension/European Society of Cardiology Hypertension Guidelines
TL;DR: The 2013 European Hypertension Guidelines are evidence based and for the first time, the level of evidence is provided for diagnostic methods and treatment recommendations, and the 2013 recommendations are updated with relevant research and recent outcome studies.
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Long‐Term Follow‐Up of Moderately Hypercholesterolemic Hypertensive Patients Following Randomization to Pravastatin vs Usual Care: The Antihypertensive and Lipid‐Lowering Treatment to Prevent Heart Attack Trial (ALLHAT‐LLT)
Karen L. Margolis,Barry R. Davis,Charles Baimbridge,Jerry O. Ciocon,Aloysius B. Cuyjet,Richard A. Dart,Paula T. Einhorn,Charles E. Ford,David Gordon,Thomas J. Hartney,L. Julian Haywood,J. L. Holtzman,David E. Mathis,Suzanne Oparil,Jeffrey L. Probstfield,Lara M. Simpson,John D. Stokes,Thomas B. Wiegmann,Jeff D. Williamson +18 more
TL;DR: Findings are consistent with evidence from other large trials that show statins prevent CHD and add evidence that they are effective for CHD prevention in black patients.
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Mechanism of renal handling of angiotensin II in the dog.
Suzanne Oparil,Michael D. Bailie +1 more
TL;DR: Observations indicate that angiotensin II is rapidly metabolized in the dog kidney by multiple enzymes, including an aminopeptidase, (2) circulating plasma enzymes do not account for the renal handling of angiotENSin II, and (3) angioten II is not sequestered in the kidney.
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The J-curve phenomenon revisited again: SPRINT outcomes favor target systolic blood pressure below 120 mmHg.
TL;DR: This animal study shows clear improvements in the ability of canine coronavirus to detect high blood pressure in mice treated with beta-blocker therapy.