Tamara Kravic-Stevovic

TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.

TL;DR: Electrochemically produced GQD irradiated with blue light generates reactive oxygen species, including singlet oxygen, and kill U251 human glioma cells by causing oxidative stress and a genetic inactivation of autophagy-essential LC3B protein partly abrogated the photodynamic cytotoxicity of G QD.

TL;DR: The results warrant caution when using compound C to inhibit AMPK-dependent cellular responses, but also support further exploration of compound C and related molecules as potential anticancer agents.

TL;DR: It is suggested that cisplatin‐triggered activation of AMPK and subsequent suppression of mTOR activity can induce an autophagic response that protects tumour cells from cisPlatin‐mediated apoptotic death.

TL;DR: AMPK-dependent protection from extracellular ASYN was also observed in rat neuron-like pheochromocytoma cell line PC12, suggesting that modulation of AMPK activity may be a promising therapeutic strategy in Parkinson's disease.