T
Tamas Revesz
Researcher at UCL Institute of Neurology
Publications - 394
Citations - 34854
Tamas Revesz is an academic researcher from UCL Institute of Neurology. The author has contributed to research in topics: Progressive supranuclear palsy & Dementia. The author has an hindex of 90, co-authored 374 publications receiving 30861 citations. Previous affiliations of Tamas Revesz include University of Cambridge & University College London.
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Journal ArticleDOI
Second consensus statement on the diagnosis of multiple system atrophy
Sid Gilman,Gregor K. Wenning,Phillip A. Low,David J. Brooks,C. J. Mathias,John Q. Trojanowski,Nicholas W. Wood,Carlo Colosimo,Alexandra Durr,Clare J. Fowler,Horacio Kaufmann,Thomas Klockgether,AJ Lees,Werner Poewe,Niall Quinn,Tamas Revesz,David Robertson,Paola Sandroni,Klaus Seppi,Marie Vidailhet +19 more
TL;DR: New criteria for diagnosis of multiple system atrophy have simplified the previous criteria, have incorporated current knowledge, and are expected to enhance future assessments of the disease.
Journal ArticleDOI
Lewy bodies in grafted neurons in subjects with Parkinson's disease suggest host-to-graft disease propagation.
Jia-Yi Li,Elisabet Englund,Janice L. Holton,Denis Soulet,Peter Hagell,Andrew J. Lees,Tammaryn Lashley,Niall Quinn,Stig Rehncrona,Anders Björklund,Håkan Widner,Tamas Revesz,Olle Lindvall,Patrik Brundin +13 more
TL;DR: Two subjects with Parkinson's disease who had long-term survival of transplanted fetal mesencephalic dopaminergic neurons (11–16 years) developed α-synuclein–positive Lewy bodies in grafted neurons, providing the first evidence, to the authors' knowledge, that the disease can propagate from host to graft cells.
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Nomenclature and nosology for neuropathologic subtypes of frontotemporal lobar degeneration: an update
Ian R. A. Mackenzie,Manuela Neumann,Eileen H. Bigio,Nigel J. Cairns,Irina Alafuzoff,Jillian J. Kril,Gabor G. Kovacs,Bernardino Ghetti,Glenda M. Halliday,Ida E. Holm,Paul G. Ince,Wouter Kamphorst,Tamas Revesz,Annemieke J.M. Rozemuller,Samir Kumar-Singh,Haruhiko Akiyama,Atik Baborie,Salvatore Spina,Dennis W. Dickson,John Q. Trojanowski,David M. A. Mann +20 more
TL;DR: A system of nosology was introduced that grouped the FTLD subtypes into broad categories, based on the molecular defect that is most characteristic, according to current evidence, and provided a concise and consistent terminology that has now been widely adopted in the literature.
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Cortical lesions in multiple sclerosis
TL;DR: Investigation of the cortical venous supply shows how such lesions may arise, and why the majority also involve the underlying white matter.
Journal ArticleDOI
A common LRRK2 mutation in idiopathic Parkinson's disease
William P. Gilks,Patrick M. Abou-Sleiman,Sonia Gandhi,Shushant Jain,Andrew B. Singleton,Andrew J. Lees,Karen Shaw,Kailash P. Bhatia,Vincenzo Bonifati,Niall Quinn,John B. Lynch,Daniel G. Healy,Janice L. Holton,Tamas Revesz,Nicholas W. Wood +14 more
TL;DR: It is shown that a common single Mendelian mutation, 2877510 g-->A, which produces a glycine to serine aminoacid substitution at codon 2019 (Gly2019 ser), in idiopathic Parkinson's disease, and suggested that testing for this mutation will be important in the management and genetic counselling of patients with Parkinson's Disease.