T
Tatiana Boyko
Researcher at Stanford University
Publications - 9
Citations - 237
Tatiana Boyko is an academic researcher from Stanford University. The author has contributed to research in topics: Wound healing & Sexual dimorphism. The author has an hindex of 4, co-authored 8 publications receiving 153 citations. Previous affiliations of Tatiana Boyko include University at Buffalo.
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Journal ArticleDOI
Review of the Current Management of Pressure Ulcers.
TL;DR: Despite an increased number of therapies available on the market, none has demonstrated any clear benefit over the others and pressure ulcer treatment remains frustrating and time-consuming.
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Laboratory Models for the Study of Normal and Pathologic Wound Healing.
TL;DR: This review aims to delineate the most relevant wound healing models currently available and to discuss their strengths and limitations in their approximation of the human wound healing processes to aid scientists in choosing the most appropriate wound healing Models for designing, testing, and validating their experiments.
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Perioperative Antibiotics in the Setting of Oropharyngeal Reconstruction: Less Is More.
Leslie E. Cohen,Brendan M. Finnerty,Alyssa R. Golas,Jill J. Ketner,Andrew L. Weinstein,Tatiana Boyko,Christine H. Rohde,David I. Kutler,Jason A. Spector +8 more
TL;DR: It is suggested that extended courses of perioperative antibiotics do not confer additional benefits in patients undergoing oropharyngeal reconstruction and a limited 48-hour course of prophylactic antibiotics with sufficient aerobic and anaerobic coverage is recommended to help minimize the incidence of antibiotic-related morbidities.
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Fabrication and In Vivo Microanastomosis of Vascularized Tissue-Engineered Constructs
Rachel C. Hooper,Karina A. Hernandez,Tatiana Boyko,Alice Harper,Jeremiah Joyce,Alyssa R. Golas,Jason A. Spector +6 more
TL;DR: This work synthesized and performed microvascular anastomosis of tissue-engineered hydrogel constructs using Pluronic(®) F127 as a sacrificial microfiber, creating microchannels within biocompatible, biodegradable type I collagen matrices.
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DEL1 protects against chondrocyte apoptosis through integrin binding.
Zhen Wang,Tatiana Boyko,Misha C. Tran,Marie F. LaRussa,Namrata Bhatia,Vania Rashidi,Michael T. Longaker,George P. Yang,George P. Yang +8 more
TL;DR: DEL1 protected chondrocytes from apoptosis in response to activators of either the intrinsic or extrinsic pathways, and to anoikis, and this effect was mediated primarily through integrin αVβ3.