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Ted Powers

Researcher at University of Minnesota

Publications -  67
Citations -  11610

Ted Powers is an academic researcher from University of Minnesota. The author has contributed to research in topics: Signal transduction & TOR complex. The author has an hindex of 39, co-authored 64 publications receiving 10044 citations. Previous affiliations of Ted Powers include University of California, San Francisco & University of California, Santa Cruz.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
Journal ArticleDOI

Regulation of Ribosome Biogenesis by the Rapamycin-sensitive TOR-signaling Pathway in Saccharomyces cerevisiae

TL;DR: Yeast control of ribosome biosynthesis by the TOR pathway is surprisingly complex and it is found that TOR signaling is a prerequisite for the induction of r-protein gene transcription that occurs in response to improved nutrient conditions.
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The TOR-controlled transcription activators GLN3, RTG1, and RTG3 are regulated in response to intracellular levels of glutamine

TL;DR: It is shown that the glutamine synthetase inhibitor l-methionine sulfoximine (MSX) specifically provokes glutamine depletion in yeast cells, suggesting that the TOR pathway senses glutamine.
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A functional pseudoknot in 16S ribosomal RNA.

TL;DR: It is demonstrated that Watson‐Crick interactions between these nucleotides are essential for ribosomal function, and certain mild perturbations of the structure generate resistance to streptomycin, an antibiotic known to interfere with the decoding process.