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Thomas G. Schnell

Researcher at Veterans Health Administration

Publications -  44
Citations -  4163

Thomas G. Schnell is an academic researcher from Veterans Health Administration. The author has contributed to research in topics: Barrett's esophagus & Esophageal disease. The author has an hindex of 23, co-authored 43 publications receiving 4086 citations. Previous affiliations of Thomas G. Schnell include United States Department of Veterans Affairs & Loyola University Chicago.

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Long-term outcome of medical and surgical therapies for gastroesophageal reflux disease: follow-up of a randomized controlled trial.

TL;DR: This study suggests that antireflux surgery should not be advised with the expectation that patients with GERD will no longer need to take antisecretory medications or that the procedure will prevent esophageal cancer among those with GERd and Barrett esophagus.
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Long-term nonsurgical management of Barrett's esophagus with high-grade dysplasia.

TL;DR: In this article, a 20-year period, patients were diagnosed and observed through an organized surveillance program at the Hines Veterans Affairs Hospital using pre-established endoscopists using preestablished endoscopic criteria.
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Five-year colon surveillance after screening colonoscopy.

TL;DR: There is a strong association between results of baseline screening colonoscopy and rate of serious incident lesions during 5.5 years of surveillance and patients with 1 or 2 tubular adenomas less than 10 mm represent a low-risk group compared with other patients with colon neoplasia.
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Most asthmatics have gastroesophageal reflux with or without bronchodilator therapy.

TL;DR: Most adult asthmatics, regardless of the use of bronchodilator therapy, have abnormal gastroesophageal reflux manifested by increased reflux frequency, delayed acid clearance during the day and night, and diminished lower esophageAL sphincter pressures.
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Hiatal hernia size, Barrett’s length, and severity of acid reflux are all risk factors for esophageal adenocarcinoma

TL;DR: High-grade dysplasia and esophageal adenocarcinoma seem to stem from an extreme and unfavorable constellation of all risk factors that are generally held responsible for the development of GERD and Barrett's esophagus.