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Tjalf Ziemssen

Researcher at Dresden University of Technology

Publications -  489
Citations -  11537

Tjalf Ziemssen is an academic researcher from Dresden University of Technology. The author has contributed to research in topics: Medicine & Multiple sclerosis. The author has an hindex of 47, co-authored 411 publications receiving 8805 citations. Previous affiliations of Tjalf Ziemssen include University of Tübingen & University of Marburg.

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Identifying prodromal Parkinson's disease: Pre-Motor disorders in Parkinson's disease

TL;DR: The evidence for the utility of olfaction, RBD, autonomic markers, visual changes, mood disorders, and cognitive loss as markers of prodromal PD and the potential sensitivity and specificity of these markers are summarized.
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Stroke-Induced Immunodepression Experimental Evidence and Clinical Relevance

TL;DR: Clinical evidence is emerging demonstrating that stroke-induced immunodepression in humans not only exists, but has very similar features to those characterized in rodent experiments.
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Association of Initial Disease-Modifying Therapy with Later Conversion to Secondary Progressive Multiple Sclerosis

TL;DR: The probability of conversion was lower when glatiramer acetate or interferon beta was started within 5 years of disease onset vs later, and initial treatment with fingolimod, alemtuzumab, or natalizumab was associated with a lower risk of conversion.
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Effector T-cell trafficking between the leptomeninges and the cerebrospinal fluid

TL;DR: The data indicate that the leptomeninges represent a checkpoint at which activated T cells are licensed to enter the CNS parenchyma and non-activated T Cells are preferentially released into the CSF, from where they can reach areas of antigen availability and tissue damage.
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Glatiramer acetate‐specific T‐helper 1‐ and 2‐type cell lines produce BDNF: implications for multiple sclerosis therapy

TL;DR: It is demonstrated that both GA-specific TH2 and TH1 cells produce the neurotrophin brain-derived neurotrophic factor (BDNF), which may be relevant for therapies with altered peptide ligands in general.