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Tobias Speerschneider
Researcher at University of Copenhagen
Publications - 16
Citations - 542
Tobias Speerschneider is an academic researcher from University of Copenhagen. The author has contributed to research in topics: Heart failure & QRS complex. The author has an hindex of 11, co-authored 16 publications receiving 464 citations. Previous affiliations of Tobias Speerschneider include National Research Foundation of South Africa & Copenhagen University Hospital.
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Journal ArticleDOI
Expression of interleukin-15 in human skeletal muscle – effect of exercise and muscle fibre type composition
Anders Rinnov Nielsen,Rémi Mounier,Peter Plomgaard,Ole Hartvig Mortensen,Milena Penkowa,Tobias Speerschneider,Henriette Pilegaard,Bente Klarlund Pedersen +7 more
TL;DR: IL‐15 mRNA level is enhanced in skeletal muscles dominated by type 2 fibres and resistance exercise induces increased muscular IL-15 mRNA levels, suggesting that muscle IL‐15 may exist in a translationally inactive pool.
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Lack of Evidence for AT1R/B2R Heterodimerization in COS-7, HEK293, and NIH3T3 Cells HOW COMMON IS THE AT1R/B2R HETERODIMER?
Jakob Lerche Hansen,Jakob Lerche Hansen,Jonas Tind Hansen,Jonas Tind Hansen,Tobias Speerschneider,Tobias Speerschneider,Christina Lyngsø,Christina Lyngsø,Niels Erikstrup,Niels Erikstrup,Ethan S. Burstein,David M. Weiner,Thomas Walther,Noriko Makita,Taroh Iiri,Nicole Merten,Evi Kostenis,Søren P. Sheikh +17 more
TL;DR: The data collectively suggest that AT1R/B2R heterodimerization does not occur as a natural consequence of their simultaneous expression in the same cell nor does the B2R influence the At1R signaling.
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Physiology and analysis of the electrocardiographic T wave in mice
TL;DR: This study seeks to make a clear definition of the murine T wave, and investigates the consequences of performing QT interval correction in anaesthetized mice.
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Sinoatrial node dysfunction induces cardiac arrhythmias in diabetic mice
TL;DR: Dysfunction of the intrinsic cardiac pacemaker and remodeling of the autonomic nervous system may conspire to increase cardiac mortality in diabetic patients, and enhanced systolic function and reduced diastolic function indicates early ventricular remodeling in obese and diabetic mice.
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Loss of K+ currents in heart failure is accentuated in KChIP2 deficient mice.
Søren Grubb,Tobias Speerschneider,Dona Occhipinti,Céline Fiset,Søren-Peter Olesen,Morten B. Thomsen,Kirstine Calloe,Kirstine Calloe +7 more
TL;DR: This work investigated remodeling associated with HF hypothesizing that KChIP2 plays a central role in the modulation of outward K+ currents in HF.