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Trevor W. Robbins

Researcher at University of Cambridge

Publications -  1184
Citations -  177352

Trevor W. Robbins is an academic researcher from University of Cambridge. The author has contributed to research in topics: Prefrontal cortex & Cognition. The author has an hindex of 231, co-authored 1137 publications receiving 164437 citations. Previous affiliations of Trevor W. Robbins include Centre national de la recherche scientifique & Massachusetts Institute of Technology.

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Effects of lesions of the nucleus basalis magnocellularis on the acquisition of cocaine self-administration in rats

TL;DR: The data suggest that the integrity of the NBM is not a critical determinant of the reinforcing effects of cocaine during the acquisition of self‐administration of the drug, but that NBM‐dependent cholinergic mechanisms may nevertheless interact with the neural substrates mediating the reinforcing properties of cocaine.
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Controlling one's world: identification of sub-regions of primate PFC underlying goal-directed behavior.

TL;DR: The role of specific prefrontal cortex (PFC) sub-regions and the caudate nucleus in mediating causal relationships between actions and their outcomes in primates are unclear.
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Serotonin depletion amplifies distinct human social emotions as a function of individual differences in personality

TL;DR: This article investigated the influence of serotonin on human emotional reactions to social conflict and found that depleting the serotonin precursor tryptophan-in a double-blind randomised placebo-controlled design-enhanced emotional responses to the scenarios in a large sample of healthy volunteers.
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Selective cholinergic denervation of the cingulate cortex impairs the acquisition and performance of a conditional visual discrimination in rats.

TL;DR: The results confirm the involvement of cholinergic innervation of the cingulate cortex in CVD performance; however, the nature of the deficit suggests a role for Cholinergic modulation in task‐relevant stimulus processing rather than stimulus‐response learning per se.
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Rule-abstraction deficits following a basal ganglia lesion.

TL;DR: The cognitive profile of a patient, PM, who had damage to the right basal ganglia as the result of a stroke was investigated, showing specific neuropsychological deficits, most notably a difficulty in developing, through ion of the relevant information, a higher-level rule by which to guide behaviour.