Trevor W. Robbins

TL;DR: Drug addiction can be understood as a pathological subversion of normal brain learning and memory processes strengthened by the motivational impact of drug-associated stimuli, leading to the establishment of compulsive drug-seeking habits.

TL;DR: It is suggested that these cognitive impairments relate to a common deficit in inhibitory control mechanisms, under the control of striatofrontal mechanisms, and that such a deficit is present in Huntington's disease mutation carriers prior to the onset of definite motor symptomatology.

TL;DR: What is currently known about the neural and psychological mechanisms of impulsivity are reviewed, and the relevance and application of these new insights to various neuropsychiatric disorders are discussed.

TL;DR: Evidence is provided for double dissociations of effects of manipulations of prefrontal cortical catecholamine and indoleamine systems that have considerable implications in the treatment of disorders such as Parkinson's disease, attention deficit/hyperactivity disorder and depression, as well as in theoretical notions of how ‘fronto-executive’ functions are subject to state-dependent influences.

TL;DR: The results suggest that 5-HT plays a critical role in regulating emotion during social decision-making and rejected a greater proportion of unfair offers, but not fair offers, without showing changes in mood, fairness judgment, basic reward processing, or response inhibition.