T
Trevor W. Robbins
Researcher at University of Cambridge
Publications - 1184
Citations - 177352
Trevor W. Robbins is an academic researcher from University of Cambridge. The author has contributed to research in topics: Prefrontal cortex & Cognition. The author has an hindex of 231, co-authored 1137 publications receiving 164437 citations. Previous affiliations of Trevor W. Robbins include Centre national de la recherche scientifique & Massachusetts Institute of Technology.
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5-HT2A and 5-HT2C receptor antagonists have opposing effects on a measure of impulsivity: interactions with global 5-HT depletion.
Catharine A. Winstanley,David E. H. Theobald,Jeffrey W. Dalley,Jeffrey C. Glennon,Trevor W. Robbins +4 more
TL;DR: Data support the view that serotonergic regulation of impulsive behaviour through different members of the 5-HT2 receptor family is functionally heterogeneous.
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Disconnection of the anterior cingulate cortex and nucleus accumbens core impairs Pavlovian approach behavior: further evidence for limbic cortical-ventral striatopallidal systems.
TL;DR: It is demonstrated that the NAcc core and Ant Cing are "nodes" of a corticostriatal circuit involved in stimulus-reward learning, and are also significantly impaired relative to controls at autoshaping.
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6-Hydroxydopamine lesions of the nucleus accumbens, but not of the caudate nucleus, attenuate enhanced responding with reward-related stimuli produced by intra-accumbens d-amphetamine.
Jane R. Taylor,Trevor W. Robbins +1 more
TL;DR: The results indicate that enhanced responding for CR following administration of psychomotor stimulant drugs is critically dependent on dopaminergic activation of the nucleus accumbens, rather than the caudate nucleus.
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The case for frontostriatal dysfunction in schizophrenia
TL;DR: A frontostriatal hypothesis of schizophrenia is advanced which takes into account evidence from studies of neurochemical pathology, functional imaging, cognitive neuropsychology, and experimental and clinical psychopharmacology, in addition to factor analyses of the symptomatic heterogeneity of this disorder.
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Tryptophan depletion in normal volunteers produces selective impairments in learning and memory.
S.B. Park,Jennifer T. Coull,Rupert McShane,Allan H. Young,Barbara J. Sahakian,Trevor W. Robbins,Philip J. Cowen +6 more
TL;DR: Computerized tests of memory, learning and executive function revealed selective and non-sedative impairments on cognitive performance following the active drink, supporting instead a specific role for the serotonergic system in the processes of memory and learning not directly implicated in frontal lobe function.