U
Ulrich Schwab
Researcher at University of Kiel
Publications - 4
Citations - 3952
Ulrich Schwab is an academic researcher from University of Kiel. The author has contributed to research in topics: Antigen & Antibody. The author has an hindex of 4, co-authored 4 publications receiving 3829 citations.
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Journal ArticleDOI
Production of a mouse monoclonal antibody reactive with a human nuclear antigen associated with cell proliferation
TL;DR: A first series of immunostainings of tumour biopsies indicated that Ki‐67 may be a potent tool for easy and quick evaluation of the proportion of proliferating cells in a tumour.
Journal ArticleDOI
Production of a monoclonal antibody specific for Hodgkin and Sternberg–Reed cells of Hodgkin's disease and a subset of normal lymphoid cells
Ulrich Schwab,Harald Stein,J. Gerdes,Hilmar Lemke,Hartmut Kirchner,Michael Schaadt,Volker Diehl +6 more
TL;DR: Production of mouse monoclonal antibodies against the Hodgkin cell line L428 was described and one hybridoma antibody was found to be specific for H and SR cells of all Hodgkin's lymphomas tested and a minute, but distinct new cell population in normal tonsils and lymph nodes.
Journal ArticleDOI
Identification of Hodgkin and Sternberg-reed cells as a unique cell type derived from a newly-detected small-cell population
Harald Stein,Johannes Gerdes,Ulrich Schwab,Hilmar Lemke,David Y. Mason,Andreas Ziegler,Wolfgang Schienle,Volker Diehl +7 more
TL;DR: It is suggested that the hitherto unknown cell population detected with the monoclonal antibody Ki‐I in normal lymphoid tissue is the normal equivalent of H and SR cells.
Journal ArticleDOI
Evidence for the detection of the normal counterpart of hodgkin and sternberg‐reed cells
Harald Stein,Johannes Gerdes,Ulrich Schwab,Hilmar Lemke,Volker Diehl,David Y. Mason,Heinrich Bartels,Andreas Ziegler +7 more
TL;DR: Frozen sections of lymph nodes from 30 patients with Hodgkin's disease were immunostained with a large panel of monoclonal antibodies reactive with cells of lymphoid tissue and granulopoiesis, showing that H and SR cells are devoid of markers specific to, or characteristic of B cells, macrophages, dendritic reticulum cells, interdigitating cells, or cells of erythropoietic or thrombopoietIC origin.