V
Valere Cacheux-Rataboul
Researcher at Genome Institute of Singapore
Publications - 9
Citations - 3504
Valere Cacheux-Rataboul is an academic researcher from Genome Institute of Singapore. The author has contributed to research in topics: Human genome & CTCF. The author has an hindex of 7, co-authored 9 publications receiving 3235 citations. Previous affiliations of Valere Cacheux-Rataboul include Agency for Science, Technology and Research.
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Journal ArticleDOI
An oestrogen-receptor-α-bound human chromatin interactome
Melissa J. Fullwood,Mei Hui Liu,You Fu Pan,Jun Liu,Han Xu,Yusoff Bin Mohamed,Yuriy L. Orlov,Stoyan Velkov,Andrea Ho,Poh Huay Mei,Elaine G.Y. Chew,Phillips Yao Hui Huang,Willem Welboren,Yuyuan Han,Hong Sain Ooi,Pramila N. Ariyaratne,Vinsensius B. Vega,Yanquan Luo,Peck Yean Tan,Pei Ye Choy,K. D. Senali Abayratna Wansa,Bing Zhao,Kar Sian Lim,Shi Chi Leow,Jit Sin Yow,Roy Joseph,Haixia Li,Kartiki V. Desai,Jane S. Thomsen,Yew Kok Lee,R. Krishna Murthy Karuturi,Thoreau Herve,Guillaume Bourque,Hendrik G. Stunnenberg,Xiaoan Ruan,Valere Cacheux-Rataboul,Wing-Kin Sung,Wing-Kin Sung,Edison T. Liu,Chia-Lin Wei,Edwin Cheung,Edwin Cheung,Edwin Cheung,Yijun Ruan,Yijun Ruan +44 more
TL;DR: It is proposed that chromatin interactions constitute a primary mechanism for regulating transcription in mammalian genomes and is described as a new strategy, chromatin interaction analysis by paired-end tag sequencing (ChIA-PET) for the de novo detection of global Chromatin interactions.
Journal ArticleDOI
CTCF-mediated functional chromatin interactome in pluripotent cells
Lusy Handoko,Han Xu,Guoliang Li,Chew Yee Ngan,Elaine G.Y. Chew,Marie Schnapp,Charlie Wah Heng Lee,Chaopeng Ye,Joanne Lim Hui Ping,Fabianus Hendriyan Mulawadi,Eleanor Wong,Eleanor Wong,Jianpeng Sheng,Yubo Zhang,Thompson Poh,Chee Seng Chan,Galih Kunarso,Atif Shahab,Guillaume Bourque,Valere Cacheux-Rataboul,Wing-Kin Sung,Wing-Kin Sung,Yijun Ruan,Chia-Lin Wei,Chia-Lin Wei,Chia-Lin Wei +25 more
TL;DR: Five distinct chromatin domains are uncovered that suggest potential new models of CTCF function in chromatin organization and transcriptional control, and demarcate chromatin-nuclear membrane attachments and influence proper gene expression through extensive cross-talk between promoters and regulatory elements.
Journal ArticleDOI
A common BIM deletion polymorphism mediates intrinsic resistance and inferior responses to tyrosine kinase inhibitors in cancer.
King Pan Ng,Axel M. Hillmer,Charles Chuah,Charles Chuah,Wen Chun Juan,Tun Kiat Ko,Audrey S.M. Teo,Pramila N. Ariyaratne,Naoto Takahashi,Kenichi Sawada,Yao Fei,Yao Fei,Sheila Soh,Wah Heng Lee,John W.J. Huang,John Carson Allen,Xing Yi Woo,Niranjan Nagarajan,Vikrant Kumar,Anbupalam Thalamuthu,Wan Ting Poh,Ai Leen Ang,Hae Tha Mya,Gee Fung How,L.Y. Yang,Liang Piu Koh,Balram Chowbay,Chia-Tien Chang,V. S. Nadarajan,Wee Joo Chng,Hein Than,Lay Cheng Lim,Yeow Tee Goh,Shenli Zhang,Dianne Poh,Patrick Tan,Patrick Tan,Ju Ee Seet,Mei-Kim Ang,Noan-Minh Chau,Quan Sing Ng,Daniel Shao-Weng Tan,Manabu Soda,Kazutoshi Isobe,Markus M. Nöthen,Tien Yin Wong,Atif Shahab,Xiaoan Ruan,Valere Cacheux-Rataboul,Wing-Kin Sung,Eng Huat Tan,Yasushi Yatabe,Hiroyuki Mano,Hiroyuki Mano,Ross A. Soo,Tan Min Chin,Wan-Teck Lim,Yijun Ruan,Yijun Ruan,S. Tiong Ong +59 more
TL;DR: The results offer an explanation for the heterogeneity of TKI responses across individuals and suggest the possibility of personalizing therapy with BH3 mimetics to overcome BIM-polymorphism–associated TKI resistance.
Journal ArticleDOI
Reprogramming of fibroblasts into induced pluripotent stem cells with orphan nuclear receptor Esrrb
Bo Feng,Jianming Jiang,Jianming Jiang,Petra Kraus,Jia-Hui Ng,Jian-Chien Dominic Heng,Jian-Chien Dominic Heng,Yun-Shen Chan,Yun-Shen Chan,Lai-Ping Yaw,Weiwei Zhang,Weiwei Zhang,Yuin-Han Loh,Yuin-Han Loh,Jianyong Han,Vinsensius B. Vega,Valere Cacheux-Rataboul,Bing Lim,Bing Lim,Thomas Lufkin,Huck-Hui Ng,Huck-Hui Ng +21 more
TL;DR: This work shows that the orphan nuclear receptor Esrrb functions in conjunction with Oct4 and Sox2 to mediate reprogramming of mouse embryonic fibroblasts (MEFs) to iPS cells, and indicates that it is possible to reprogram MEFs without exogenous Klf transcription factors and link a nuclear receptor to somatic cell reprograming.
Journal ArticleDOI
Comprehensive long-span paired-end-tag mapping reveals characteristic patterns of structural variations in epithelial cancer genomes.
Axel M. Hillmer,Fei Yao,Fei Yao,Koichiro Inaki,Wah Heng Lee,Pramila N. Ariyaratne,Audrey S.M. Teo,Xing Yi Woo,Zhenshui Zhang,Hao Zhao,Leena Ukil,Jieqi P. Chen,Feng Zhu,Jimmy Bok Yan So,Manuel Salto-Tellez,Wan Ting Poh,Kelson F B Zawack,Niranjan Nagarajan,Song Gao,Guoliang Li,Vikrant Kumar,Hui Ping J Lim,Yee Yen Sia,Chee Seng Chan,See Ting Leong,Say Chuan Neo,Poh Sum D Choi,Hervé Thoreau,Patrick Tan,Patrick Tan,Atif Shahab,Xiaoan Ruan,Jonas Bergh,Per Hall,Valere Cacheux-Rataboul,Chia-Lin Wei,Khay Guan Yeoh,Wing-Kin Sung,Guillaume Bourque,Edison T. Liu,Yijun Ruan,Yijun Ruan +41 more
TL;DR: The quantitative and connective nature of DNA-PET data is precise in delineating the genealogy of complex rearrangement events, and it is discovered that large duplications are among the initial rearrangements that trigger genome instability for extensive amplification in epithelial cancers.