V
Vaqar M. Adhami
Researcher at University of Wisconsin-Madison
Publications - 119
Citations - 11402
Vaqar M. Adhami is an academic researcher from University of Wisconsin-Madison. The author has contributed to research in topics: Cancer & Prostate cancer. The author has an hindex of 63, co-authored 118 publications receiving 10484 citations.
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Journal ArticleDOI
Targeting Microtubules by Natural Agents for Cancer Therapy
TL;DR: A review of the successful cancer chemotherapy from natural compounds in clinical settings and the challenges that may abort their usefulness, including resistance to the existing MTAs.
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Pomegranate fruit juice for chemoprevention and chemotherapy of prostate cancer
TL;DR: It is suggested that pomegranate juice may have cancer-chemopreventive as well as cancer-chemotherapeutic effects against prostate cancer in humans.
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Introducing nanochemoprevention as a novel approach for cancer control: proof of principle with green tea polyphenol epigallocatechin-3-gallate.
Imtiaz A. Siddiqui,Vaqar M. Adhami,Dhruba J. Bharali,Bilal Bin Hafeez,Mohammad Asim,Sabih I. Khwaja,Nihal Ahmad,Huadong Cui,Shaker A. Mousa,Hasan Mukhtar +9 more
TL;DR: This study encapsulated green tea polyphenol epigallocatechin-3-gallate (EGCG) in polylactic acid-polyethylene glycol nanoparticles and observed that encapsulated EGCG retains its biological effectiveness with over 10-fold dose advantage for exerting its proapoptotic and angiogenesis inhibitory effects, critically important determinants of chemopreventive effects of E GCG in both in vitro and in vivo systems.
Journal Article
Resveratrol causes WAF-1/p21-mediated G(1)-phase arrest of cell cycle and induction of apoptosis in human epidermoid carcinoma A431 cells.
TL;DR: This is the first systematic study showing the involvement of each component of cyclin-dependent kinase (cdk) inhibitor-cyclin-cdk machinery during cell cycle arrest and apoptosis of cancer cells by resveratrol.
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Oral consumption of green tea polyphenols inhibits insulin-like growth factor-I-induced signaling in an autochthonous mouse model of prostate cancer.
TL;DR: It is suggested that IGF-I/IGFBP-3 signaling pathway is a prime pathway for green tea polyphenol-mediated inhibition of prostate cancer that limits the progression of cancer through inhibition of angiogenesis and metastasis.