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Wei Qiu

Researcher at Sun Yat-sen University

Publications -  256
Citations -  5756

Wei Qiu is an academic researcher from Sun Yat-sen University. The author has contributed to research in topics: Neuromyelitis optica & Medicine. The author has an hindex of 33, co-authored 214 publications receiving 4356 citations. Previous affiliations of Wei Qiu include University of Western Australia & Loyola University Chicago.

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International consensus diagnostic criteria for neuromyelitis optica spectrum disordersAuthor Response

TL;DR: Criteria for neuromyelitis optica (NMO) spectrum disorders (NMOSD) was proposed that incorporated unifying the terms NMO and NMOSD, including patients with aquaporin-4 (AQP4)–immunoglobulin G (IgG) negative/unknown, thus giving these patients …
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PUMA Regulates Intestinal Progenitor Cell Radiosensitivity and Gastrointestinal Syndrome

TL;DR: A critical role of the BH3-only protein p53 upregulated modulator of apoptosis (PUMA) in the radiosensitivity of intestinal epithelium and pathogenesis of GI syndrome is reported here and PUMA-mediated apoptosis in the progenitor and stem cell compartments is crucial for radiation-induced intestinal damage.
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PUMA-mediated intestinal epithelial apoptosis contributes to ulcerative colitis in humans and mice

TL;DR: PUMA induction contributes to the pathogenesis of colitis by promoting IEC apoptosis and suggest that PUMA inhibition may be an effective strategy to promote mucosal healing in patients with UC.
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Interleukin-17-secreting T cells in neuromyelitis optica and multiple sclerosis during relapse.

TL;DR: IL-17-secreting CD8(+)T cells were increased in patients with NMO and MS during relapse and have an important role in the pathological mechanism of N MO and MS.
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PUMA is directly activated by NF-κB and contributes to TNF-α-induced apoptosis

TL;DR: It is found that p53 upregulated modulator of apoptosis (PUMA), a p53 downstream target and a BH3-only Bcl-2 family member, is directly regulated by NF-κB in response to TNF-α.