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William A. Maher
Researcher at University of Canberra
Publications - 271
Citations - 8723
William A. Maher is an academic researcher from University of Canberra. The author has contributed to research in topics: Arsenic & Arsenobetaine. The author has an hindex of 46, co-authored 267 publications receiving 7985 citations. Previous affiliations of William A. Maher include Applied Science Private University & Cooperative Research Centre.
Papers
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Journal ArticleDOI
Biotransference and biomagnification of selenium copper, cadmium, zinc, arsenic and lead in a temperate seagrass ecosystem from Lake Macquarie Estuary, NSW, Australia
M Barwick,William A. Maher +1 more
TL;DR: Selenium was found to biomagnify, exceeding maximum permitted concentrations for human consumption within carnivorous fish tissue, the highest trophic level examined, and arsenic showed some evidence of biomagnification.
Journal ArticleDOI
Invertebrate biomarkers: links to toxicosis that predict population decline
Ross V Hyne,William A. Maher +1 more
TL;DR: Evaluated biochemical biomarkers of impaired biological function in invertebrates should not be used as a replacement for conventional aquatic monitoring techniques, but should be applied as supplementary approaches for demonstrating links between sublethal biochemical and adverse effects in natural populations in field studies.
MonographDOI
Ecological Assessment of Selenium in the Aquatic Environment
Peter M. Chapman,William J. Adams,Marjorie L. Brooks,Charles G. Delos,Samuel N. Luoma,William A. Maher,Harry M. Ohlendorf,Theresa S. Presser,Patrick Shaw +8 more
TL;DR: In this article, the authors present the context for selenium risk assessment in the context of a global problem, which they call Selenium risk assessment (SRL) problem.
Handbook for sediment quality assessment
Journal ArticleDOI
Toxicity, biotransformation, and mode of action of arsenic in two freshwater microalgae (Chlorella sp. and Monoraphidium arcuatum)
Jacqueline L. Levy,Jacqueline L. Levy,Jennifer L. Stauber,Merrin S. Adams,William A. Maher,Jason K. Kirby,Dianne F. Jolley +6 more
TL;DR: Arsenic toxicity most likely was due to arsenite accumulation in the cell, when the ability to excrete and/or methylate arsenite was overwhelmed at high arsenic concentrations.