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Xin Yang

Bio: Xin Yang is an academic researcher from Northeast Forestry University. The author has contributed to research in topics: Apoptosis & Oxidative stress. The author has an hindex of 4, co-authored 7 publications receiving 75 citations.

Papers
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TL;DR: The results highlighted the need to take precautions against copper and arsenic co-exposure when considering their impact in susceptible animals/populations and can function independently or cooperatively to affect oxidative stress, mitochondrial dynamics and programmed cell death.

35 citations

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TL;DR: The results unambiguously suggested that under arsenic stress, zinc can partly relieve intestinal inflammation and disruption of tight junction segment-dependently.

25 citations

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TL;DR: The results revealed that increased apoptosis and autophagy levels induced by Cu2+ and arsenite appeared to be independent of oxidative stress, which didn't have significant changes in different treatment groups at the same time point, and the redox balance gradually deviated with the extension of time.

15 citations

Journal ArticleDOI
Xin Yang, Lu Yao, Yu Wang, Xinran Zhang, Pengfei Ren 
TL;DR: In this article , peroxymonosulfate (PMS) was tested for its ability to improve Fe(III) coagulation of Microcystis aeruginosa in water treatment.

14 citations

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TL;DR: These findings provide a basic understanding of mitochondrial abnormality-initiated neuropathology in response to environmental pollutant mixtures, suggesting an adaptive response to the frangibility of the central nerve system.

11 citations


Cited by
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Journal ArticleDOI
Qi Han1, Jingyang Zhang1, Qi Sun1, Yanmin Xu1, Xiaohua Teng1 
TL;DR: It is concluded that NH3 caused-oxidative stress and mitochondrial dysfunction mediated nephrocyte necroptosis in chickens and may provide new insight into NH3 nephrotoxicity.

72 citations

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TL;DR: The results proved that folic acid and starch decoration were increased the NPs penetration in cell through folate receptor-based endocytosis for enhanced breast cancer therapy.

70 citations

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TL;DR: This review discusses different aspects of the use of metal chelators, alone or in combination, in the treatment of As poisoning, and considers the possible role of flavonoids, trace elements, and herbal drugs as promising natural chelating and detoxifying agents.
Abstract: Arsenic (As) is widely used in the modern industry, especially in the production of pesticides, herbicides, wood preservatives, and semiconductors. The sources of As such as contaminated water, air, soil, but also food, can cause serious human diseases. The complex mechanism of As toxicity in the human body is associated with the generation of free radicals and the induction of oxidative damage in the cell. One effective strategy in reducing the toxic effects of As is the usage of chelating agents, which provide the formation of inert chelator-metal complexes with their further excretion from the body. This review discusses different aspects of the use of metal chelators, alone or in combination, in the treatment of As poisoning. Consideration is given to the therapeutic effect of thiol chelators such as meso-2,3-dimercaptosuccinic acid, sodium 2,3-dimercapto-1-propanesulfonate, 2,3-dimercaptopropanol, penicillamine, ethylenediaminetetraacetic acid, and other recent agents against As toxicity. The review also considers the possible role of flavonoids, trace elements, and herbal drugs as promising natural chelating and detoxifying agents.

61 citations

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TL;DR: Findings provide strong evidence that excess Cu can trigger autophagy and apoptosis via the mitochondrial pathway by inducing oxidative stress in rat kidneys.

58 citations

Journal ArticleDOI
Yu Wang1, Hongjing Zhao1, Mengyao Mu1, Menghao Guo1, Mingwei Xing1 
TL;DR: Treatment with Zn appears to offer protection against As-induced splenic injury in common carp and the pathologic changes above were alleviated, elucidating the role of Zn, a natural immune-modulator, as a potential antidote against As poisoning.

54 citations