scispace - formally typeset
Y

Ya Cao

Researcher at Central South University

Publications -  399
Citations -  12057

Ya Cao is an academic researcher from Central South University. The author has contributed to research in topics: Nasopharyngeal carcinoma & Cancer. The author has an hindex of 52, co-authored 276 publications receiving 8809 citations. Previous affiliations of Ya Cao include Chinese Ministry of Education & National Health and Family Planning Commission.

Papers
More filters
Journal ArticleDOI

Tumor-Associated Neutrophils Recruit Macrophages and T-Regulatory Cells to Promote Progression of Hepatocellular Carcinoma and Resistance to Sorafenib.

TL;DR: TAN-conditioned media, as well as recombinant CCL2 and CCL17, increased the migratory activity of the macrophages and T-regulatory cells from patients or mice with HCC to a greater extent that PBN- conditioned media.
Journal ArticleDOI

Emerging roles of lipid metabolism in cancer metastasis

TL;DR: The present review is focused on key enzymes in lipid metabolism associated with metastatic disease pathogenesis and the function of an important membrane structure-lipid raft in mediating tumor aggressive progression.
Journal ArticleDOI

Long noncoding RNA LINC00336 inhibits ferroptosis in lung cancer by functioning as a competing endogenous RNA

TL;DR: It is demonstrated that a nuclear lncRNA LINC00336 is upregulated in lung cancer and functions as an oncogene by acting as a competing endogenous RNA (ceRNAs) and found that MIR6852 inhibited cell growth by promoting ferroptosis.
Journal ArticleDOI

A G3BP1-Interacting lncRNA Promotes Ferroptosis and Apoptosis in Cancer via Nuclear Sequestration of p53.

TL;DR: It is demonstrated that the cytosolic lncRNA P53RRA is downregulated in cancers and functions as a tumor suppressor by inhibiting cancer progression, thus regulating p53 modulators to suppress cancer progression.
Journal ArticleDOI

EGLN1/c-Myc Induced Lymphoid-Specific Helicase Inhibits Ferroptosis through Lipid Metabolic Gene Expression Changes

TL;DR: The molecular basis of the c-Myc/EGLN1-mediated induction of LSH expression that inhibits ferroptosis is elucidated, which can be exploited for the development of therapeutic strategies targeting ferroPTosis for the treatment of cancer.