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Yasushi Kitaoka

Researcher at St. Marianna University School of Medicine

Publications -  67
Citations -  7802

Yasushi Kitaoka is an academic researcher from St. Marianna University School of Medicine. The author has contributed to research in topics: Optic nerve & Retinal ganglion cell. The author has an hindex of 22, co-authored 63 publications receiving 6333 citations. Previous affiliations of Yasushi Kitaoka include University of Southern California.

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Guidelines for the use and interpretation of assays for monitoring autophagy (3rd edition)

Daniel J. Klionsky, +2522 more
- 21 Jan 2016 - 
TL;DR: In this paper, the authors present a set of guidelines for the selection and interpretation of methods for use by investigators who aim to examine macro-autophagy and related processes, as well as for reviewers who need to provide realistic and reasonable critiques of papers that are focused on these processes.
Journal ArticleDOI

Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)

Daniel J. Klionsky, +2983 more
- 08 Feb 2021 - 
TL;DR: In this article, the authors present a set of guidelines for investigators to select and interpret methods to examine autophagy and related processes, and for reviewers to provide realistic and reasonable critiques of reports that are focused on these processes.
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TNF-α-Induced Optic Nerve Degeneration and Nuclear Factor-κB p65

TL;DR: Inhibition of NF-kappaB p65 with AS ODN or helenalin significantly ameliorated the effects of TNF-alpha-mediated axonal loss, with the possible involvement of microglial cells.
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Involvement of RhoA and possible neuroprotective effect of fasudil, a Rho kinase inhibitor, in NMDA-induced neurotoxicity in the rat retina.

TL;DR: The results suggest that RhoA and ROCKII are upregulated and may be involved in NMDA-induced retinal neurotoxicity, and that fasudil is neuroprotective against glutamate-related excitotoxicity.
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Axonal protection by Nmnat3 overexpression with involvement of autophagy in optic nerve degeneration

TL;DR: The results suggest that the axonal-protective effect of Nmnat3 may be involved in autophagy machinery, and that modulation of N Mnat3 and Autophagy may lead to potential strategies against degenerative optic nerve disease.