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Yehudith Naot
Researcher at Technion – Israel Institute of Technology
Publications - 18
Citations - 2117
Yehudith Naot is an academic researcher from Technion – Israel Institute of Technology. The author has contributed to research in topics: Mycoplasma pulmonis & Gene. The author has an hindex of 12, co-authored 18 publications receiving 2010 citations.
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Molecular Biology and Pathogenicity of Mycoplasmas
TL;DR: There is now solid genetic support for the hypothesis that mycoplasmas have evolved as a branch of gram-positive bacteria by a process of reductive evolution and developed various genetic systems providing a highly plastic set of variable surface proteins to evade the host immune system.
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Subversion and exploitation of host cells by mycoplasmas
Shlomo Rottem,Yehudith Naot +1 more
TL;DR: Mycoplasmas are minute wall-less bacterial parasites that exhibit strict host and tissue specificities and modulate the activity of host cells by a variety of direct mechanisms and/or indirectly by cytokine-mediated effects.
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Identification of human sperm antigens reacting with antisperm antibodies from sera and genital tract secretions
Shafrira Shai,Yehudith Naot +1 more
TL;DR: Sperm antigens exhibiting relative molecular weights of 62 kd were identified as the major sperm antIGens reacting with antibodies present in seminal plasmas from infertile males, which differ from the antigen recognized by local antisperm antibodies.
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Mitogenicity and Pathogenicity of Mycoplasma pulmonis in Rats. I. Atypical Interstitial Pneumonia Induced by Mitogenic Mycoplasmal Membranes
TL;DR: The results suggest that activation of thymus-derived lymphocytes is the major cause of the pneumonia resulting from infections with M. pulmonis, and indicate a correlation of mitogenicity and pathogenicity.
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Role of Gene 46 in Bacteriophage T4 Deoxyribonucleic Acid Synthesis
Channa Shalitin,Yehudith Naot +1 more
TL;DR: In an attempt to establish whether Escherichia coli B infected with N130 (an amber mutant defective in gene 46) is recombination-deficient, the postinfection fate of (14)C-labeled N130 parental deoxyribonucleic acid (DNA) was followed, its amount in complex with the host cell membrane being determined in sucrose gradients after mild lysis of the infected cells.