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Yi Wang

Researcher at Peking University

Publications -  9
Citations -  120

Yi Wang is an academic researcher from Peking University. The author has contributed to research in topics: Glucagon & Insulin. The author has an hindex of 4, co-authored 8 publications receiving 45 citations. Previous affiliations of Yi Wang include Huazhong University of Science and Technology.

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Metabolic stress drives sympathetic neuropathy within the liver.

TL;DR: In this paper, the authors examined neural distributions in the mouse, nonhuman primate, and human livers with advanced 3D imaging and found that neural innervations within the liver are predominantly sympathetic, but not parasympathetic, inputs.
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Progress of the development of reactors for pyrolysis of municipal waste

TL;DR: In this article, the authors revisited the progress of the investigation of the characteristics of the pyrolysis of municipal waste in different types of reactors, especially in terms of the kinetics and the distribution of products.
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A unified deep-learning network to accurately segment insulin granules of different animal models imaged under different electron microscopy methodologies.

TL;DR: To automatically and precisely segment the insulin granules with different sizes and pattern features in EM images, the authors built and trained a multi-branch fully convolutional network (MFCN) and collected three-dimensional images of pancreatic beta cells in wild type and MS rhesus monkeys with a FIB-SEM and manually annotated granules from a relatively small number of images.
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Sarm1-mediated neurodegeneration within the enteric nervous system protects against local inflammation of the colon.

TL;DR: In this paper, Sarm1-mediated axonal degeneration within the enteric nervous system (ENS) of the mouse, non-human primate, and human by advanced 3D imaging was observed.
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An optical method to evaluate both mass and functional competence of pancreatic α- and β-cells.

TL;DR: The initial glucose intolerance that appears in 10-week-old db/db mice is associated with further expansion of α- cell mass prior to deterioration in functional β-cell mass, which shall benefit mechanistic studies of imbalanced hormone secretion during type 2 diabetes progression.