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Ying Liu

Researcher at Shandong University

Publications -  13
Citations -  238

Ying Liu is an academic researcher from Shandong University. The author has contributed to research in topics: Chemistry & Medicine. The author has an hindex of 6, co-authored 7 publications receiving 149 citations.

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Atorvastatin ameliorates experimental autoimmune neuritis by decreased Th1/Th17 cytokines and up-regulated T regulatory cells.

TL;DR: There is strong evidence that atorvastatin can act as an inhibitor in EAN by inhibiting the immune response of Th1 and Th17, decreasing the expression of co-stimulatory molecule, and up-regulating the number of T regulatory cells.
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M1 Macrophage Derived Exosomes Aggravate Experimental Autoimmune Neuritis via Modulating Th1 Response

TL;DR: A previously undescribed means that M1 macrophages amplify Th1 response via exosomes is identified and novel insights into the crosstalk between macrophage and T cells as well are provided.
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Atorvastatin-modified dendritic cells in vitro ameliorate experimental autoimmune myasthenia gravis by up-regulated Treg cells and shifted Th1/Th17 to Th2 cytokines

TL;DR: This study found that spleen-derived dendritic cells from the ongoing experimental autoimmune myasthenia gravis (EAMG) rats can be induced into tolerogenic DCs by atorvastatin in vitro, and played their immunomodulatory effects in vivo mainly by decreased expression of CD86 and MHC class II on endogenous DCs.
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A retrospective case series of segmental zoster paresis of limbs: clinical, electrophysiological and imaging characteristics.

TL;DR: SZP is associated with obvious limb weakness, nerve axons lesions and localization to nerve roots, plexus or peripheral nerves, according to electromyography findings and MRI scans in patients with motor weakness after zoster infection.
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Dynamic intracellular exchange of nanomaterials’ protein corona perturbs proteostasis and remodels cell metabolism

TL;DR: It was revealed that PKM2 depletion was causative to CMA-induced cell metabolism disruption from glycolysis to lipid metabolism, and chaperone-mediated autophagy activity in cells.