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Yoshiaki Masuyama

Researcher at Wakayama Medical University

Publications -  135
Citations -  1228

Yoshiaki Masuyama is an academic researcher from Wakayama Medical University. The author has contributed to research in topics: Norepinephrine (medication) & Essential hypertension. The author has an hindex of 16, co-authored 135 publications receiving 1212 citations.

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Bone mineral density in women with essential hypertension.

TL;DR: The results of the present study demonstrated that DXA provided an index of whole calcium balance, and suggest that high blood pressure might be associated with reduced BMD in female hypertension.
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Nitric oxide improves membrane fluidity of erythrocytes in essential hypertension: An electron paramagnetic resonance investigation.

TL;DR: The greater effect of NO on the fluidity in essential hypertension suggests that NO might actively participate in the regulation of rheological behavior of erythrocytes and have a crucial role in the improvement of microcirculation in hypertension.
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Electron Paramagnetic Resonance Investigation on Modulatory Effect of 17β-Estradiol on Membrane Fluidity of Erythrocytes in Postmenopausal Women

TL;DR: E2 significantly increased the membrane fluidity and improved the microviscosity of erythrocyte membranes, partially mediated by an NO- and cGMP-dependent pathway and might have a crucial role in the improvement of the microcirculation in hypertension.
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Effects of galanin on dopamine release in the central nervous system of normotensive and spontaneously hypertensive rats.

TL;DR: Results show that galanin significantly reduced the release of dopamine in rat striatum, and the greater inhibitory effect of galan in on dopamine release in SHR suggests that galansin might actively participate in the regulation of dopaminergic nerve activity in hypertension.
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Enhanced DNA synthesis of cultured vascular smooth muscle cells from spontaneously hypertensive rats. Difference of response to growth factor, intracellular free calcium concentration and DNA synthesizing cell cycle.

TL;DR: It is suggested that enhanced DNA synthesis in CVSMCs from spontaneously hypertensive rats (SHR) is determined genetically, thatEnhanced DNA synthesis is largely dependent on an increased proportion of S-phase cells and that this increase in S- phase cells in CVDs from SHR could be due to enhanced competence gene expression in SHR cells.