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Zhishuang Wang

Other affiliations: Chinese Ministry of Education
Bio: Zhishuang Wang is an academic researcher from Tianjin University of Technology. The author has contributed to research in topics: Population & Epidemic model. The author has an hindex of 5, co-authored 7 publications receiving 273 citations. Previous affiliations of Zhishuang Wang include Chinese Ministry of Education.

Papers
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Journal ArticleDOI
TL;DR: This paper investigates the interaction between the disease transmission and disease-related awareness spread, and proposes a new coupled disease spreading model on a two-layered multiplex network, where one layer denotes the underlying topology for the epidemics and the other one represents the corresponding topologies for the awareness spread.

152 citations

Journal ArticleDOI
TL;DR: A novel epidemic model is proposed by using two-layer multiplex networks to investigate the multiple influence between awareness diffusion and epidemic propagation, where the upper layer represents the awareness diffusion regarding epidemics and the lower layer expresses the epidemic propagation.

140 citations

Journal ArticleDOI
TL;DR: The microscopic Markov chain (MMC) approach is analytically derived the expression of the epidemic threshold for the proposed epidemic model, which indicates that the diffusion of positive and negative prevention information, as well as the topology of the physical contact network have a significant impact on the epidemic thresholds.
Abstract: We propose a novel epidemic model based on two-layered multiplex networks to explore the influence of positive and negative preventive information on epidemic propagation. In the model, one layer represents a social network with positive and negative preventive information spreading competitively, while the other one denotes the physical contact network with epidemic propagation. The individuals who are aware of positive prevention will take more effective measures to avoid being infected than those who are aware of negative prevention. Taking the microscopic Markov chain (MMC) approach, we analytically derive the expression of the epidemic threshold for the proposed epidemic model, which indicates that the diffusion of positive and negative prevention information, as well as the topology of the physical contact network have a significant impact on the epidemic threshold. By comparing the results obtained with MMC and those with the Monte Carlo (MC) simulations, it is found that they are in good agreement, but MMC can well describe the dynamics of the proposed model. Meanwhile, through extensive simulations, we demonstrate the impact of positive and negative preventive information on the epidemic threshold, as well as the prevalence of infectious diseases. We also find that the epidemic prevalence and the epidemic outbreaks can be suppressed by the diffusion of positive preventive information and be promoted by the diffusion of negative preventive information.

128 citations

Journal ArticleDOI
TL;DR: The experimental results demonstrate that accelerating positive information dissemination and enhancing the propaganda intensity of mass media can efficaciously restrain the epidemicSpread of negative information can also alleviate the epidemic to a certain extent when the positive information hardly spreads.
Abstract: During epidemic outbreaks, there are various types of information about epidemic prevention disseminated simultaneously among the population. Meanwhile, the mass media also scrambles to report the information related to the epidemic. Inspired by these phenomena, we devise a model to discuss the dynamical characteristics of the co-evolution spreading of multiple information and epidemic under the influence of mass media. We construct the co-evolution model under the framework of two-layered networks and gain the dynamical equations and epidemic critical point with the help of the micro-Markov chain approach. The expression of epidemic critical point show that the positive and negative information have a direct impact on the epidemic critical point. Moreover, the mass media can indirectly affect the epidemic size and epidemic critical point through their interference with the dissemination of epidemic-relevant information. Though extensive numerical experiments, we examine the accuracy of the dynamical equations and expression of the epidemic critical point, showing that the dynamical characteristics of co-evolution spreading can be well described by the dynamic equations and the epidemic critical point is able to be accurately calculated by the derived expression. The experimental results demonstrate that accelerating positive information dissemination and enhancing the propaganda intensity of mass media can efficaciously restrain the epidemic spreading. Interestingly, the way to accelerate the dissemination of negative information can also alleviate the epidemic to a certain extent when the positive information hardly spreads. Current results can provide some useful clues for epidemic prevention and control on the basis of epidemic-relevant information dissemination.

36 citations

Journal ArticleDOI
TL;DR: A novel two-city epidemic model is proposed, where the spreading process is depicted by using SIR (susceptible-infected-recovered) model, and the results indicate that they are globally asymptotic stable.

29 citations


Cited by
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Proceedings ArticleDOI
22 Jan 2006
TL;DR: Some of the major results in random graphs and some of the more challenging open problems are reviewed, including those related to the WWW.
Abstract: We will review some of the major results in random graphs and some of the more challenging open problems. We will cover algorithmic and structural questions. We will touch on newer models, including those related to the WWW.

7,116 citations

Journal ArticleDOI
TL;DR: A novel epidemic model is proposed by using two-layer multiplex networks to investigate the multiple influence between awareness diffusion and epidemic propagation, where the upper layer represents the awareness diffusion regarding epidemics and the lower layer expresses the epidemic propagation.

140 citations

Posted Content
TL;DR: In this paper, the authors introduce the simple game model into the interdependent networks composed of two networks, and show that when the inter-dependent factor is smaller than a particular value, homogeneous cooperation can be guaranteed.
Abstract: Spatial evolution game has traditionally assumed that players interact with neighbors on a single network, which is isolated and not influenced by other systems. We introduce the simple game model into the interdependent networks composed of two networks, and show that when the interdependent factor $\alpha$ is smaller than a particular value $\alpha_C$, homogeneous cooperation can be guaranteed. However, as interdependent factor exceeds $\alpha_C$, spontaneous symmetry breaking of fraction of cooperators presents itself between different networks. In addition, our results can be well predicted by the strategy-couple pair approximation method.

136 citations

Journal Article
TL;DR: This work develops a framework for understanding the robustness of interacting networks subject to cascading failures and presents exact analytical solutions for the critical fraction of nodes that, on removal, will lead to a failure cascade and to a complete fragmentation of two interdependent networks.
Abstract: Complex networks have been studied intensively for a decade, but research still focuses on the limited case of a single, non-interacting network. Modern systems are coupled together and therefore should be modelled as interdependent networks. A fundamental property of interdependent networks is that failure of nodes in one network may lead to failure of dependent nodes in other networks. This may happen recursively and can lead to a cascade of failures. In fact, a failure of a very small fraction of nodes in one network may lead to the complete fragmentation of a system of several interdependent networks. A dramatic real-world example of a cascade of failures (‘concurrent malfunction’) is the electrical blackout that affected much of Italy on 28 September 2003: the shutdown of power stations directly led to the failure of nodes in the Internet communication network, which in turn caused further breakdown of power stations. Here we develop a framework for understanding the robustness of interacting networks subject to such cascading failures. We present exact analytical solutions for the critical fraction of nodes that, on removal, will lead to a failure cascade and to a complete fragmentation of two interdependent networks. Surprisingly, a broader degree distribution increases the vulnerability of interdependent networks to random failure, which is opposite to how a single network behaves. Our findings highlight the need to consider interdependent network properties in designing robust networks.

132 citations

Journal ArticleDOI
TL;DR: The microscopic Markov chain (MMC) approach is analytically derived the expression of the epidemic threshold for the proposed epidemic model, which indicates that the diffusion of positive and negative prevention information, as well as the topology of the physical contact network have a significant impact on the epidemic thresholds.
Abstract: We propose a novel epidemic model based on two-layered multiplex networks to explore the influence of positive and negative preventive information on epidemic propagation. In the model, one layer represents a social network with positive and negative preventive information spreading competitively, while the other one denotes the physical contact network with epidemic propagation. The individuals who are aware of positive prevention will take more effective measures to avoid being infected than those who are aware of negative prevention. Taking the microscopic Markov chain (MMC) approach, we analytically derive the expression of the epidemic threshold for the proposed epidemic model, which indicates that the diffusion of positive and negative prevention information, as well as the topology of the physical contact network have a significant impact on the epidemic threshold. By comparing the results obtained with MMC and those with the Monte Carlo (MC) simulations, it is found that they are in good agreement, but MMC can well describe the dynamics of the proposed model. Meanwhile, through extensive simulations, we demonstrate the impact of positive and negative preventive information on the epidemic threshold, as well as the prevalence of infectious diseases. We also find that the epidemic prevalence and the epidemic outbreaks can be suppressed by the diffusion of positive preventive information and be promoted by the diffusion of negative preventive information.

128 citations