Example of Cell Biology and Toxicology format
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Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format
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Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format Example of Cell Biology and Toxicology format
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open access Open Access
recommended Recommended

Cell Biology and Toxicology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Toxicology #9 of 122 up up by 9 ranks
Health, Toxicology and Mutagenesis #18 of 134 up up by 2 ranks
Cell Biology #45 of 279 up up by 56 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 139 Published Papers | 1269 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 09/07/2020
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FAQ

Related Journals

open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 9.6
SJR: 1.264
SNIP: 1.419
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 12.0
SJR: 1.748
SNIP: 1.959

Multidisciplinary Digital Publishing Institute

Quality:  
High
CiteRatio: 5.5
SJR: 1.047
SNIP: 1.291
open access Open Access
recommended Recommended

Elsevier

Quality:  
High
CiteRatio: 10.8
SJR: 2.136
SNIP: 1.846

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

6.284

23% from 2018

Impact factor for Cell Biology and Toxicology from 2016 - 2019
Year Value
2019 6.284
2018 5.097
2017 3.39
2016 2.333
graph view Graph view
table view Table view

9.1

5% from 2019

CiteRatio for Cell Biology and Toxicology from 2016 - 2020
Year Value
2020 9.1
2019 8.7
2018 6.9
2017 6.1
2016 4.6
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 23% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 5% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

0.842

31% from 2019

SJR for Cell Biology and Toxicology from 2016 - 2020
Year Value
2020 0.842
2019 1.216
2018 0.908
2017 0.924
2016 0.769
graph view Graph view
table view Table view

0.9

7% from 2019

SNIP for Cell Biology and Toxicology from 2016 - 2020
Year Value
2020 0.9
2019 0.966
2018 0.807
2017 0.706
2016 0.938
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 31% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has decreased by 7% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Cell Biology and Toxicology

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Springer

Cell Biology and Toxicology

Cell Biology and Toxicology is an international journal which provides a rapid publication outlet for papers of high scientific standards in the areas of cell biology, genetic, molecular and cellular toxicology.The scope of publication includes scientific reports dealing with ...... Read More

Health, Toxicology and Mutagenesis

Cell Biology

Environmental Science

i
Last updated on
09 Jul 2020
i
ISSN
0742-2091
i
Impact Factor
Medium - 0.833
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

Journal Article DOI: 10.1007/S10565-005-0085-6
The Caco-2 cell line as a model of the intestinal barrier: influence of cell and culture-related factors on Caco-2 cell functional characteristics
Yula Sambuy, I. De Angelis1, Giulia Ranaldi, Maria Laura Scarino, Annalaura Stammati1, F. Zucco

Abstract:

The human intestinal Caco-2 cell line has been extensively used over the last twenty years as a model of the intestinal barrier. The parental cell line, originally obtained from a human colon adenocarcinoma, undergoes in culture a process of spontaneous differentiation that leads to the formation of a monolayer of cells, expr... The human intestinal Caco-2 cell line has been extensively used over the last twenty years as a model of the intestinal barrier. The parental cell line, originally obtained from a human colon adenocarcinoma, undergoes in culture a process of spontaneous differentiation that leads to the formation of a monolayer of cells, expressing several morphological and functional characteristics of the mature enterocyte. Culture-related conditions were shown to influence the expression of these characteristics, in part due to the intrinsic heterogeneity of the parental cell line, leading to selection of sub-populations of cells becoming prominent in the culture. In addition, several clonal cell lines have been isolated from the parental line, exhibiting in general a more homogeneous expression of differentiation traits, while not always expressing all characteristics of the parental line. Culture-related conditions, as well as the different Caco-2 cell lines utilized in different laboratories, often make it extremely difficult to compare results in the literature. This review is aimed at summarizing recent, or previously unreviewed, data from the literature on the effects of culture-related factors and the influence of line sub-types (parental vs. different clonal lines) on the expression of differentiation traits important for the use of Caco-2 cells as a model of the absorptive and defensive properties of the intestinal mucosa. Since the use of Caco-2 cells has grown exponentially in recent years, it is particularly important to highlight these methodological aspects in order to promote the standardization and optimisation of this intestinal model. read more read less

Topics:

Intestinal mucosa (61%)61% related to the paper, Cell culture (52%)52% related to the paper
1,253 Citations
open accessOpen access Journal Article DOI: 10.1007/S10565-011-9208-4
Characterization of primary human hepatocytes, HepG2 cells, and HepaRG cells at the mRNA level and CYP activity in response to inducers and their predictivity for the detection of human hepatotoxins.
UCB1

Abstract:

In the pharmaceutical industry, improving the early detection of drug-induced hepatotoxicity is essential as it is one of the most important reasons for attrition of candidate drugs during the later stages of drug development. The first objective of this study was to better characterize different cellular models (i.e., HepG2,... In the pharmaceutical industry, improving the early detection of drug-induced hepatotoxicity is essential as it is one of the most important reasons for attrition of candidate drugs during the later stages of drug development. The first objective of this study was to better characterize different cellular models (i.e., HepG2, HepaRG cells, and fresh primary human hepatocytes) at the gene expression level and analyze their metabolic cytochrome P450 capabilities. The cellular models were exposed to three different CYP450 inducers; beta-naphthoflavone (BNF), phenobarbital (PB), and rifampicin (RIF). HepG2 cells responded very weakly to the different inducers at the gene expression level, and this translated generally into low CYP450 activities in the induced cells compared with the control cells. On the contrary, HepaRG cells and the three human donors were inducible after exposure to BNF, PB, and RIF according to gene expression responses and CYP450 activities. Consequently, HepaRG cells could be used in screening as a substitute and/or in complement to primary hepatocytes for CYP induction studies. The second objective was to investigate the predictivity of the different cellular models to detect hepatotoxins (16 hepatotoxic and 5 nonhepatotoxic compounds). Specificity was 100% with the different cellular models tested. Cryopreserved human hepatocytes gave the highest sensitivity, ranging from 31% to 44% (depending on the donor), followed by lower sensitivity (13%) for HepaRG and HepG2 cells (6.3%). Overall, none of the models under study gave desirable sensitivities (80–100%). Consequently, a high metabolic capacity and CYP inducibility in cell lines does not necessarily correlate with a high sensitivity for the detection of hepatotoxic drugs. Further investigations are necessary to compare different cellular models and determine those that are best suited for the detection of hepatotoxic compounds. read more read less
View PDF
557 Citations
Journal Article DOI: 10.1007/BF00756522
The human intestinal epithelial cell line Caco-2; pharmacological and pharmacokinetic applications.
V Meunier, M. Bourrié, Y Berger, G Fabre

Abstract:

The gastrointestinal tract remains the most popular and acceptable route of administration for drugs. It offers the great advantage of convenience and many compounds are well absorbed and thereby provide acceptable plasma concentration-time profiles. Currently there is considerable interest from the pharmaceutical industry in... The gastrointestinal tract remains the most popular and acceptable route of administration for drugs. It offers the great advantage of convenience and many compounds are well absorbed and thereby provide acceptable plasma concentration-time profiles. Currently there is considerable interest from the pharmaceutical industry in development of cell culture systems that would mimic the intestinal mucosa in order to evaluate strategies for investigating and/or enhancing drug absorption. The intestinal epithelial cells of primary interest, from the standpoint of drug absorption and metabolism, are the villus cells, which are fully differentiated cells. An in vitro cell culture system consisting of a monolayer of viable, polarized and fully differentiated villus cells, similar to that found in the small intestine, would be a valuable tool in the study of drug and nutrient transport and metabolism. The Caco-2 cell line, which exhibits a well-differentiated brush border on the apical surface and tight junctions, and expresses typical small-intestinal microvillus hydrolases and nutrient transporters, has proven to be the most popular in vitro model (a) to rapidly assess the cellular permeability of potential drug candidates, (b) to elucidate pathways of drug transport (e.g., passive versus carrier mediated), (c) to assess formulation strategies designed to enhance membrane permeability, (d) to determine the optimal physicochemical characteristics for passive diffusion of drugs, and (e) to assess potential toxic effects of drug candidates or formulation components on this biological barrier. Since differentiated Caco-2 cells express various cytochrome P450 isoforms and phase II enzymes such as UDP-glucuronosyltransferases, sulfotransferases and glutathione-S-transferases, this model could also allow the study of presystemic drug metabolism. read more read less

Topics:

Intestinal mucosa (57%)57% related to the paper, Membrane permeability (56%)56% related to the paper, Drug metabolism (54%)54% related to the paper, Biological activity (51%)51% related to the paper, Caco-2 (50%)50% related to the paper
394 Citations
Journal Article DOI: 10.1007/S10565-006-0140-Y
Adriamycin-induced oxidative mitochondrial cardiotoxicity
Jessica Berthiaume1, Kendall B. Wallace1

Abstract:

The anticancer agent Adriamycin (ADR) has long been recognized to induce a dose-limiting cardiotoxicity. Numerous studies have attempted to characterize and elucidate the mechanism(s) behind its cardiotoxic effect. Despite a wealth of data covering a wide-range of effects mediated by the drug, the definitive mechanism remains... The anticancer agent Adriamycin (ADR) has long been recognized to induce a dose-limiting cardiotoxicity. Numerous studies have attempted to characterize and elucidate the mechanism(s) behind its cardiotoxic effect. Despite a wealth of data covering a wide-range of effects mediated by the drug, the definitive mechanism remains a matter of debate. However, there is consensus that this toxicity is related to the induction of reactive oxygen species (ROS). Induction of ROS in the heart by ADR occurs via redox cycling of the drug at complex I of the electron transport chain. Many studies support the theory that mitochondria are a primary target of ADR-induced oxidative stress, both acutely and long-term. This review focuses on the effects of ADR redox cycling on the mitochondrion, which support the hypothesis that these organelles are indeed a major factor in ADR cardiotoxicity. This review has been constructed with particular emphasis on studies utilizing cardiac models with clinically relevant doses or concentrations of ADR in the hope of advancing our understanding of the mechanisms of ADR toxicity. This compilation of current data may reveal valuable insights for the development of therapeutic strategies better tailored to minimizing the dose-limiting effect of ADR. read more read less

Topics:

Cardiotoxicity (53%)53% related to the paper, Heart metabolism (51%)51% related to the paper
389 Citations
Journal Article DOI: 10.1007/BF00755791
A microtiter plate assay for total glutathione and glutathione disulfide contents in cultured/isolated cells: performance study of a new miniaturized protocol.
C Vandeputte1, I Guizon1, I Genestie-Denis1, B Vannier1, G. Lorenzon1

Abstract:

The microtiter plate technique reported by Baker and colleagues for the glutathione reductase-DTNB recycling assay of total glutathione (GSx) and glutathione disulfide (GSSG) has been modified according to Anderson's recommendations, in order to improve the reliability and accuracy of this miniaturized method for the measurem... The microtiter plate technique reported by Baker and colleagues for the glutathione reductase-DTNB recycling assay of total glutathione (GSx) and glutathione disulfide (GSSG) has been modified according to Anderson's recommendations, in order to improve the reliability and accuracy of this miniaturized method for the measurement of glutathione status in cultured/isolated cells. Dilute HCl (10 mmol/L) has been used to lyse cells, before protein removal by centrifugation in the presence of 1.3% sulfosalicylic acid. The final DTNB, GSSG-reductase and NADPH concentrations in the reaction mixture have been increased to 0.7 mmol/L, 1.2 IU/ml and 0.24 mmol/L, respectively. The procedure specificity has been tested by spiking and dilution assays, showing that about 90% of the expected GSx amounts could actually be recovered, while no changes of GSSG concentrations were caused in the cells. Accuracy has been assessed by analysis of within-series precision as well as of intra- and interassay reproducibility, showing coefficient variation of < 10%. Glutathione changes measured either in control rat hepatocytes or in primary cultures treated with paracetamol or menadione were in good agreement with well-known literature data. These data suggest that the experimental conditions reported in this paper are suitable for the analysis of total glutathione and glutathione disulfide concentrations in cultured/isolated cells. read more read less

Topics:

Glutathione disulfide (66%)66% related to the paper, Glutathione (58%)58% related to the paper, Sulfosalicylic acid (55%)55% related to the paper, Microtiter plate (51%)51% related to the paper
351 Citations
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Cell Biology and Toxicology format uses SPBASIC citation style.

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Frequently asked questions

1. Can I write Cell Biology and Toxicology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Cell Biology and Toxicology guidelines and auto format it.

2. Do you follow the Cell Biology and Toxicology guidelines?

Yes, the template is compliant with the Cell Biology and Toxicology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Cell Biology and Toxicology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Cell Biology and Toxicology citation style.

4. Can I use the Cell Biology and Toxicology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Cell Biology and Toxicology.

5. Can I use a manuscript in Cell Biology and Toxicology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Cell Biology and Toxicology that you can download at the end.

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It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Cell Biology and Toxicology.

7. Where can I find the template for the Cell Biology and Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Biology and Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Cell Biology and Toxicology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

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SciSpace's Cell Biology and Toxicology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Cell Biology and Toxicology?

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After writing your paper autoformatting in Cell Biology and Toxicology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Cell Biology and Toxicology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Cell Biology and Toxicology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Cell Biology and Toxicology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Cell Biology and Toxicology?

The 5 most common citation types in order of usage for Cell Biology and Toxicology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Cell Biology and Toxicology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Cell Biology and Toxicology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Cell Biology and Toxicology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Cell Biology and Toxicology Endnote style according to Elsevier guidelines.

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