Example of Glia format
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Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format Example of Glia format
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This content is only for preview purposes. The original open access content can be found here.
open access Open Access
recommended Recommended

Glia — Template for authors

Publisher: Wiley
Categories Rank Trend in last 3 yrs
Neurology #8 of 156 down down by 2 ranks
Cellular and Molecular Neuroscience #8 of 88 down down by 1 rank
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 639 Published Papers | 6813 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 10/07/2020
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Related Journals

open access Open Access

Springer

Quality:  
High
CiteRatio: 6.0
SJR: 1.239
SNIP: 1.096
open access Open Access

Springer

Quality:  
High
CiteRatio: 6.8
SJR: 1.582
SNIP: 1.194
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 10.1
SJR: 2.615
SNIP: 1.649
open access Open Access
recommended Recommended

Springer

Quality:  
High
CiteRatio: 9.9
SJR: 1.569
SNIP: 1.158

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

5.984

3% from 2018

Impact factor for Glia from 2016 - 2019
Year Value
2019 5.984
2018 5.829
2017 5.846
2016 6.2
graph view Graph view
table view Table view

10.7

7% from 2019

CiteRatio for Glia from 2016 - 2020
Year Value
2020 10.7
2019 10.0
2018 10.2
2017 11.3
2016 11.8
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 3% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 7% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.954

8% from 2019

SJR for Glia from 2016 - 2020
Year Value
2020 2.954
2019 2.73
2018 2.855
2017 3.318
2016 3.598
graph view Graph view
table view Table view

1.449

13% from 2019

SNIP for Glia from 2016 - 2020
Year Value
2020 1.449
2019 1.288
2018 1.358
2017 1.309
2016 1.362
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 8% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 13% in last years.
  • This journal’s SNIP is in the top 10 percentile category.
Glia

Guideline source: View

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Wiley

Glia

GLIA is a peer-reviewed journal which publishes articles dealing with all aspects of glial structure and function. This includes anatomy, physiology, biochemistry, pharmacology, immunology, and pathology of glial cells.... Read More

Neuroscience

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Last updated on
10 Jul 2020
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ISSN
0894-1491
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Impact Factor
High - 1.41
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Open Access
Yes
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Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
apa
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Citation Type
Author Year
(Blonder et al., 1982)
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Bibliography Example
Blonder, G. E., Tinkham, M., & Klapwijk, T. M. (1982). Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B, 25(7), 4515–4532.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1002/GLIA.20467
Systemic LPS Causes Chronic Neuroinflammation and Progressive Neurodegeneration
01 Apr 2007 - Glia

Abstract:

Inflammation is implicated in the progressive nature of neurodegenerative diseases, such as Parkinson's disease, but the mechanisms are poorly understood. A single systemic lipopolysaccharide (LPS, 5 mg/kg, i.p.) or tumor necrosis factor alpha (TNFα, 0.25 mg/kg, i.p.) injection was administered in adult wild-type mice and in ... Inflammation is implicated in the progressive nature of neurodegenerative diseases, such as Parkinson's disease, but the mechanisms are poorly understood. A single systemic lipopolysaccharide (LPS, 5 mg/kg, i.p.) or tumor necrosis factor alpha (TNFα, 0.25 mg/kg, i.p.) injection was administered in adult wild-type mice and in mice lacking TNFα receptors (TNF R1/R2−/−) to discern the mechanisms of inflammation transfer from the periphery to the brain and the neurodegenerative consequences. Systemic LPS administration resulted in rapid brain TNFα increase that remained elevated for 10 months, while peripheral TNFα (serum and liver) had subsided by 9 h (serum) and 1 week (liver). Systemic TNFα and LPS administration activated microglia and increased expression of brain pro-inflammatory factors (i.e., TNFα, MCP-1, IL-1β, and NF-κB p65) in wild-type mice, but not in TNF R1/R2−/− mice. Further, LPS reduced the number of tyrosine hydroxylase-immunoreactive neurons in the substantia nigra (SN) by 23% at 7-months post-treatment, which progressed to 47% at 10 months. Together, these data demonstrate that through TNFα, peripheral inflammation in adult animals can: (1) activate brain microglia to produce chronically elevated pro-inflammatory factors; (2) induce delayed and progressive loss of DA neurons in the SN. These findings provide valuable insight into the potential pathogenesis and self-propelling nature of Parkinson's disease. read more read less

Topics:

Neuroinflammation (56%)56% related to the paper, Neurodegeneration (53%)53% related to the paper, Tumor necrosis factor alpha (52%)52% related to the paper, Microglia (52%)52% related to the paper, Inflammation (51%)51% related to the paper
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1,802 Citations
open accessOpen access Journal Article DOI: 10.1002/GLIA.10161
Microglia as a source and target of cytokines.
Uwe-Karsten Hanisch1
01 Nov 2002 - Glia

Abstract:

Cytokines constitute a significant portion of the immuno- and neuromodulatory messengers that can be released by activated microglia. By virtue of potent effects on resident and invading cells, microglial cyto- and chemokines regulate innate defense mechanisms, help the initiation and influence the type of immune responses, p... Cytokines constitute a significant portion of the immuno- and neuromodulatory messengers that can be released by activated microglia. By virtue of potent effects on resident and invading cells, microglial cyto- and chemokines regulate innate defense mechanisms, help the initiation and influence the type of immune responses, participate in the recruitment of leukocytes to the CNS, and support attempts of tissue repair and recovery. Microglia can also receive cyto- and chemokine signals as part of auto- and paracrine communications with astrocytes, neurons, the endothelium, and leukocyte infiltrates. Strong responses and modulatory influences can be demonstrated, adding to the emerging view that microglial behavior is highly dependent on the (cytokine) environment and that reactions to a challenge may vary with the stimulation context. In principle, microglial activation aims at CNS protection. However, failed microglial engagement due to excessive or sustained activation could significantly contribute to acute and chronic neuropathologies. Dysregulation of microglial cytokine production could thereby promote harmful actions of the defense mechanisms, result in direct neurotoxicity, as well as disturb neural cell functions as they are sensitive to cytokine signaling. read more read less

Topics:

Microglia (52%)52% related to the paper, Cytokine (52%)52% related to the paper, Neuroglia (52%)52% related to the paper, Chemokine (50%)50% related to the paper
View PDF
1,508 Citations
Journal Article DOI: 10.1002/GLIA.20207
Astrocyte activation and reactive gliosis
Milos Pekny1, Michael Nilsson1
01 Jun 2005 - Glia

Abstract:

Astrocytes become activated (reactive) in response to many CNS pathologies, such as stroke, trauma, growth of a tumor, or neurodegenerative disease. The process of astrocyte activation remains rather enigmatic and results in so-called "reactive gliosis," a reaction with specific structural and functional characteristics. In s... Astrocytes become activated (reactive) in response to many CNS pathologies, such as stroke, trauma, growth of a tumor, or neurodegenerative disease. The process of astrocyte activation remains rather enigmatic and results in so-called "reactive gliosis," a reaction with specific structural and functional characteristics. In stroke or in CNS trauma, the lesion itself, the ischemic environment, disrupted blood-brain barrier, the inflammatory response, as well as in metabolic, excitotoxic, and in some cases oxidative crises--all affect the extent and quality of reactive gliosis. The fact that astrocytes function as a syncytium of interconnected cells both in health and in disease, rather than as individual cells, adds yet another dimension to this picture. This review focuses on several aspects of astrocyte activation and reactive gliosis and discusses its possible roles in the CNS trauma and ischemia. Particular emphasis is placed on the lessons learnt from mouse genetic models in which the absence of intermediate filament proteins in astrocytes leads to attenuation of reactive gliosis with distinct pathophysiological and clinical consequences. read more read less

Topics:

Gliosis (61%)61% related to the paper, Neuroglia (53%)53% related to the paper, Genetic model (53%)53% related to the paper, Astrocyte (50%)50% related to the paper
1,492 Citations
Journal Article DOI: 10.1002/GLIA.1106
Immune function of microglia
Francesca Aloisi1
01 Nov 2001 - Glia

Abstract:

During the past decade, mechanisms involved in the immune surveillance of the central nervous system (CNS) have moved to the forefront of neuropathological research mainly because of the recognition that most neurological disorders involve activation and, possibly, dysregulation of microglia, the intrinsic macrophages of the ... During the past decade, mechanisms involved in the immune surveillance of the central nervous system (CNS) have moved to the forefront of neuropathological research mainly because of the recognition that most neurological disorders involve activation and, possibly, dysregulation of microglia, the intrinsic macrophages of the CNS. Increasing evidence indicates that, in addition to their well-established phagocytic function, microglia may also participate in the regulation of non specific inflammation as well as adaptive immune responses. This article focuses on the signals regulating microglia innate immune functions, the role of microglia in antigen presentation, and their possible involvement in the development of CNS immunopathology. read more read less

Topics:

Neuroinflammation (63%)63% related to the paper, Neuroglia (54%)54% related to the paper, Innate immune system (54%)54% related to the paper, Microglia (53%)53% related to the paper, Immune system (51%)51% related to the paper
1,241 Citations
Journal Article DOI: 10.1002/GLIA.1107
Immune function of astrocytes.
Yuanshu Dong1, Etty N. Benveniste1
01 Nov 2001 - Glia

Abstract:

Astrocytes are the major glial cell within the central nervous system (CNS) and have a number of important physiological properties related to CNS homeostasis. The aspect of astrocyte biology addressed in this review article is the astrocyte as an immunocompetent cell within the brain. The capacity of astrocytes to express cl... Astrocytes are the major glial cell within the central nervous system (CNS) and have a number of important physiological properties related to CNS homeostasis. The aspect of astrocyte biology addressed in this review article is the astrocyte as an immunocompetent cell within the brain. The capacity of astrocytes to express class II major histocompatibility complex (MHC) antigens and costimulatory molecules (B7 and CD40) that are critical for antigen presentation and T-cell activation are discussed. The functional role of astrocytes as immune effector cells and how this may influence aspects of inflammation and immune reactivity within the brain follows, emphasizing the involvement of astrocytes in promoting Th2 responses. The ability of astrocytes to produce a wide array of chemokines and cytokines is discussed, with an emphasis on the immunological properties of these mediators. The significance of astrocytic antigen presentation and chemokine/cytokine production to neurological diseases with an immunological component is described. read more read less

Topics:

Neuroglia (59%)59% related to the paper, Antigen presentation (55%)55% related to the paper, Astrocyte (54%)54% related to the paper, Major histocompatibility complex (53%)53% related to the paper, Immune system (52%)52% related to the paper
1,220 Citations
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SciSpace is a very innovative solution to the formatting problem and existing providers, such as Mendeley or Word did not really evolve in recent years.

- Andreas Frutiger, Researcher, ETH Zurich, Institute for Biomedical Engineering

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What to expect from SciSpace?

Speed and accuracy over MS Word

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With SciSpace, you do not need a word template for Glia.

It automatically formats your research paper to Wiley formatting guidelines and citation style.

You can download a submission ready research paper in pdf, LaTeX and docx formats.

Time comparison

Time taken to format a paper and Compliance with guidelines

Plagiarism Reports via Turnitin

SciSpace has partnered with Turnitin, the leading provider of Plagiarism Check software.

Using this service, researchers can compare submissions against more than 170 million scholarly articles, a database of 70+ billion current and archived web pages. How Turnitin Integration works?

Turnitin Stats
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Easy support from all your favorite tools

Glia format uses apa citation style.

Automatically format and order your citations and bibliography in a click.

SciSpace allows imports from all reference managers like Mendeley, Zotero, Endnote, Google Scholar etc.

Frequently asked questions

1. Can I write Glia in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Glia guidelines and auto format it.

2. Do you follow the Glia guidelines?

Yes, the template is compliant with the Glia guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Glia?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Glia citation style.

4. Can I use the Glia templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Glia.

5. Can I use a manuscript in Glia that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Glia that you can download at the end.

6. How long does it usually take you to format my papers in Glia?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Glia.

7. Where can I find the template for the Glia?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Glia's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Glia's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Glia an online tool or is there a desktop version?

SciSpace's Glia is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Glia?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Glia?”

11. What is the output that I would get after using Glia?

After writing your paper autoformatting in Glia, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Glia's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Glia?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Glia. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Glia?

The 5 most common citation types in order of usage for Glia are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Glia?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Glia's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Glia in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Glia Endnote style according to Elsevier guidelines.

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I spent hours with MS word for reformatting. It was frustrating - plain and simple. With SciSpace, I can draft my manuscripts and once it is finished I can just submit. In case, I have to submit to another journal it is really just a button click instead of an afternoon of reformatting.

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