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Journal of Virology — Template for authors

Publisher: American Society for Microbiology

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Guideline source

Categories	Rank	Trend in last 3 yrs
Insect Science	#2 of 153	-
Microbiology	#21 of 150	down by 6 ranks
Virology	#13 of 69	down by 2 ranks
Immunology	#41 of 202	down by 9 ranks

Journal quality:

High

Last 4 years overview: 3160 Published Papers | 28053 Citations

Indexed in: Scopus

Last updated: 28/06/2020

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Insights

General info

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PLOS Pathogens

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Quality:

High

CiteRatio: 11.0

SJR: 3.719

SNIP: 1.882

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Cellular Microbiology

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Quality:

Good

CiteRatio: 6.5

SJR: 1.542

SNIP: 0.992

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Nature Microbiology

Nature

Quality:

High

CiteRatio: 28.2

SJR: 7.305

SNIP: 3.41

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Frontiers in Cellular and Infection Microbiology

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Quality:

High

CiteRatio: 6.5

SJR: 1.812

SNIP: 1.485

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

4.501

4% from 2018

Impact factor for Journal of Virology from 2016 - 2019
Year	Value
2019	4.501
2018	4.324
2017	4.368
2016	4.663

Graph view

8.9

13% from 2019

CiteRatio for Journal of Virology from 2016 - 2020
Year	Value
2020	8.9
2019	7.9
2018	8.4
2017	9.0
2016	9.2

Graph view

Insights

Impact factor of this journal has increased by 4% in last year.
This journal’s impact factor is in the top 10 percentile category.

Insights

CiteRatio of this journal has increased by 13% in last years.
This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

2.617

9% from 2019

SJR for Journal of Virology from 2016 - 2020
Year	Value
2020	2.617
2019	2.406
2018	2.59
2017	2.853
2016	3.114

Graph view

1.372

27% from 2019

SNIP for Journal of Virology from 2016 - 2020
Year	Value
2020	1.372
2019	1.084
2018	1.074
2017	1.106
2016	1.123

Graph view

Insights

SJR of this journal has increased by 9% in last years.
This journal’s SJR is in the top 10 percentile category.

Insights

SNIP of this journal has increased by 27% in last years.
This journal’s SNIP is in the top 10 percentile category.

Journal of Virology

Guideline source: View

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American Society for Microbiology

Journal of Virology

Journal of Virology (JVI) explores the nature of viruses, reporting important new discoveries and pointing to new directions in research. Original research articles cover the viruses of animals, archaea, bacteria, fungi, plants, and protozoa. Among the key issues investigated are virus structure and assembly, viral genome replication and regulation of viral gene expression, viral genetic diversity and evolution, virus-cell interactions, cellular responses to viral infection, transformation and oncogenesis, gene delivery, viral pathogenesis and immunity, and vaccines and antiviral agents. Read Less

Insect Science

Virology

Microbiology

Immunology

Agricultural and Biological Sciences

Last updated on	28 Jun 2020
ISSN	0022-538X
Impact Factor	High - 1.255
Open Access	Yes
Sherpa RoMEO Archiving Policy	Green
Plagiarism Check	Available via Turnitin
Endnote Style	Download Available
Bibliography Name	unsrt asm custom citation
Citation Type	Numbered (25)
Bibliography Example	Blonder, G. E., Tinkham, M., and Klapwijk, T. M. 1982. Transition from metallic to tunneling regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B, 25(7):4515–4532.

Top papers written in this journal

Open access • Journal Article • DOI: 10.1128/JVI.00127-20 •

Receptor Recognition by the Novel Coronavirus from Wuhan: an Analysis Based on Decade-Long Structural Studies of SARS Coronavirus.

Yushun Wan¹, Jian Shang¹, •

17 Mar 2020 - Journal of Virology

Abstract:

Recently, a novel coronavirus (2019-nCoV) has emerged from Wuhan, China, causing symptoms in humans similar to those caused by severe acute respiratory syndrome coronavirus (SARS-CoV). Since the SARS-CoV outbreak in 2002, extensive structural analyses have revealed key atomic-level interactions between the SARS-CoV spike prot... Recently, a novel coronavirus (2019-nCoV) has emerged from Wuhan, China, causing symptoms in humans similar to those caused by severe acute respiratory syndrome coronavirus (SARS-CoV). Since the SARS-CoV outbreak in 2002, extensive structural analyses have revealed key atomic-level interactions between the SARS-CoV spike protein receptor-binding domain (RBD) and its host receptor angiotensin-converting enzyme 2 (ACE2), which regulate both the cross-species and human-to-human transmissions of SARS-CoV. Here, we analyzed the potential receptor usage by 2019-nCoV, based on the rich knowledge about SARS-CoV and the newly released sequence of 2019-nCoV. First, the sequence of 2019-nCoV RBD, including its receptor-binding motif (RBM) that directly contacts ACE2, is similar to that of SARS-CoV, strongly suggesting that 2019-nCoV uses ACE2 as its receptor. Second, several critical residues in 2019-nCoV RBM (particularly Gln493) provide favorable interactions with human ACE2, consistent with 2019-nCoV's capacity for human cell infection. Third, several other critical residues in 2019-nCoV RBM (particularly Asn501) are compatible with, but not ideal for, binding human ACE2, suggesting that 2019-nCoV has acquired some capacity for human-to-human transmission. Last, while phylogenetic analysis indicates a bat origin of 2019-nCoV, 2019-nCoV also potentially recognizes ACE2 from a diversity of animal species (except mice and rats), implicating these animal species as possible intermediate hosts or animal models for 2019-nCoV infections. These analyses provide insights into the receptor usage, cell entry, host cell infectivity and animal origin of 2019-nCoV and may help epidemic surveillance and preventive measures against 2019-nCoV.IMPORTANCE The recent emergence of Wuhan coronavirus (2019-nCoV) puts the world on alert. 2019-nCoV is reminiscent of the SARS-CoV outbreak in 2002 to 2003. Our decade-long structural studies on the receptor recognition by SARS-CoV have identified key interactions between SARS-CoV spike protein and its host receptor angiotensin-converting enzyme 2 (ACE2), which regulate both the cross-species and human-to-human transmissions of SARS-CoV. One of the goals of SARS-CoV research was to build an atomic-level iterative framework of virus-receptor interactions to facilitate epidemic surveillance, predict species-specific receptor usage, and identify potential animal hosts and animal models of viruses. Based on the sequence of 2019-nCoV spike protein, we apply this predictive framework to provide novel insights into the receptor usage and likely host range of 2019-nCoV. This study provides a robust test of this reiterative framework, providing the basic, translational, and public health research communities with predictive insights that may help study and battle this novel 2019-nCoV. read more

Topics:

Coronavirus (55%)55% related to the paper

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3,527 Citations

Open access • Journal Article • DOI: 10.1128/JVI.72.11.8463-8471.1998 •

A Third-Generation Lentivirus Vector with a Conditional Packaging System

Tom Dull, Romain Zufferey¹, •

01 Nov 1998 - Journal of Virology

Abstract:

Vectors derived from human immunodeficiency virus (HIV) are highly efficient vehicles for in vivo gene delivery. However, their biosafety is of major concern. Here we exploit the complexity of the HIV genome to provide lentivirus vectors with novel biosafety features. In addition to the structural genes, HIV contains two regu... Vectors derived from human immunodeficiency virus (HIV) are highly efficient vehicles for in vivo gene delivery. However, their biosafety is of major concern. Here we exploit the complexity of the HIV genome to provide lentivirus vectors with novel biosafety features. In addition to the structural genes, HIV contains two regulatory genes, tat and rev, that are essential for HIV replication, and four accessory genes that encode critical virulence factors. We previously reported that the HIV type 1 accessory open reading frames are dispensable for efficient gene transduction by a lentivirus vector. We now demonstrate that the requirement for the tat gene can be offset by placing constitutive promoters upstream of the vector transcript. Vectors generated from constructs containing such a chimeric long terminal repeat (LTR) transduced neurons in vivo at very high efficiency, whether or not they were produced in the presence of Tat. When the rev gene was also deleted from the packaging construct, expression of gag and pol was strictly dependent on Rev complementation in trans. By the combined use of a separate nonoverlapping Rev expression plasmid and a 5' LTR chimeric transfer construct, we achieved optimal yields of vector of high transducing efficiency (up to 10(7) transducing units [TU]/ml and 10(4) TU/ng of p24). This third-generation lentivirus vector uses only a fractional set of HIV genes: gag, pol, and rev. Moreover, the HIV-derived constructs, and any recombinant between them, are contingent on upstream elements and trans complementation for expression and thus are nonfunctional outside of the vector producer cells. This split-genome, conditional packaging system is based on existing viral sequences and acts as a built-in device against the generation of productive recombinants. While the actual biosafety of the vector will ultimately be proven in vivo, the improved design presented here should facilitate testing of lentivirus vectors. read more

Topics:

Expression vector (54%)54% related to the paper, Lentivirus (54%)54% related to the paper, Transduction (genetics) (51%)51% related to the paper,

3,063 Citations

Open access • Journal Article • DOI: 10.1128/JVI.59.2.284-291.1986 •

Production of acquired immunodeficiency syndrome-associated retrovirus in human and nonhuman cells transfected with an infectious molecular clone.

Akio Adachi, Howard E. Gendelman,

01 Aug 1986 - Journal of Virology

Abstract:

We constructed an infectious molecular clone of acquired immunodeficiency syndrome-associated retrovirus. Upon transfection, this clone directed the production of infectious virus particles in a wide variety of cells in addition to human T4 cells. The progeny, infectious virions, were synthesized in mouse, mink, monkey, and s... We constructed an infectious molecular clone of acquired immunodeficiency syndrome-associated retrovirus. Upon transfection, this clone directed the production of infectious virus particles in a wide variety of cells in addition to human T4 cells. The progeny, infectious virions, were synthesized in mouse, mink, monkey, and several human non-T cell lines, indicating the absence of any intracellular obstacle to viral RNA or protein production or assembly. During the course of these studies, a human colon carcinoma cell line, exquisitely sensitive to DNA transfection, was identified. read more

Topics:

Clone (cell biology) (59%)59% related to the paper, Human Immunodeficiency Virus Proteins (58%)58% related to the paper, Retrovirus (56%)56% related to the paper,

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2,894 Citations

Open access • Journal Article • DOI: 10.1128/JVI.68.7.4650-4655.1994 •

Temporal association of cellular immune responses with the initial control of viremia in primary human immunodeficiency virus type 1 syndrome.

Richard A. Koup¹, Jeffrey T. Safrit¹, •

01 Jul 1994 - Journal of Virology

Abstract:

Virologic and immunologic studies were performed on five patients presenting with primary human immunodeficiency virus type 1 (HIV-1) infection. CD8+ cytotoxic T lymphocyte (CTL) precursors specific for cells expressing antigens of HIV-1 Gag, Pol, and Env were detected at or within 3 weeks of presentation in four of the five ... Virologic and immunologic studies were performed on five patients presenting with primary human immunodeficiency virus type 1 (HIV-1) infection. CD8+ cytotoxic T lymphocyte (CTL) precursors specific for cells expressing antigens of HIV-1 Gag, Pol, and Env were detected at or within 3 weeks of presentation in four of the five patients and were detected in all five patients by 3 to 6 months after presentation. The one patient with an absent initial CTL response had prolonged symptoms, persistent viremia, and low CD4+ T-cell count. Neutralizing antibody activity was absent at the time of presentation in all five patients. These findings suggest that cellular immunity is involved in the initial control of virus replication in primary HIV-1 infection and indicate a role for CTL in protective immunity to HIV-1 in vivo. read more

Topics:

CTL* (59%)59% related to the paper, Cellular immunity (58%)58% related to the paper, Viremia (55%)55% related to the paper,

2,614 Citations

Open access • Journal Article • DOI: 10.1128/JVI.73.1.152-160.1999 •

Full-Length Human Immunodeficiency Virus Type 1 Genomes from Subtype C-Infected Seroconverters in India, with Evidence of Intersubtype Recombination

Kavita S. Lole¹, Robert C. Bollinger, •

01 Jan 1999 - Journal of Virology

Abstract:

According to World Health Organization estimates, India will have the greatest number of human immunodeficiency virus (HIV)-infected individuals of any country by the end of this decade (1, 6). High rates of sexually transmitted diseases, rapidly increasing seroprevalence in female commercial sex workers, and inadequate facil... According to World Health Organization estimates, India will have the greatest number of human immunodeficiency virus (HIV)-infected individuals of any country by the end of this decade (1, 6). High rates of sexually transmitted diseases, rapidly increasing seroprevalence in female commercial sex workers, and inadequate facilities for HIV testing, counseling, and prevention are the major contributing factors in the recent explosive increases in the numbers of HIV infections (5, 6, 24, 29). While antiretroviral drugs have reduced mortality from AIDS in developed nations, their effect will be negligible elsewhere due to their cost. For most communicable diseases, vaccines offer the most cost-effective control strategy. It is likely that development of a vaccine for HIV will require knowledge of the viral variants being transmitted in the target population. Despite India’s impending predominance in the worldwide pandemic, little is known of the genetic diversity of HIV-1 in India. The HIV-1 sequence database is growing exponentially, but the distribution of submitted sequences is not representative of the worldwide picture. Subtype C has been reported in nearly every region affected by HIV-1 (11, 23, 28) and predominates in India, and it also causes 74% of infections in southern Africa and 96% of infections in northern Africa (11, 18, 32). Given the combined population of India and the other regions affected, subtype C is likely to be the most commonly transmitted HIV-1 subtype worldwide. In contrast, 7% of the available HIV-1 sequence data is from subtype C-infected individuals (37), and of the 46 completely sequenced HIV-1 genomes (excluding multiple derivatives of HIV-1LAI), only two are of subtype C, one from a 1992 Brazilian sample and the other from a 1986 Ethiopian sample (37). In November 1997, an analysis of cross-clade epitope variation (9) excluded the C clade from evaluation of p24gag epitopes because of a lack of sequence data, whereas there was sufficient data to analyze subtypes A, B, D, F, G, and H (no HIV-1 harboring a subtype E gag gene has been found). Further sequence data from subtype C is needed, but the past approach of generating data from small subgenomic amplicons is no longer sufficient. Recent developments have made full-genome characterization of HIV-1 isolates both important and feasible. First, the recognition of intersubtype recombination in a significant proportion of HIV-1 sequences (44, 45) has led to detection of mosaic genomes in many regions of the world affected by multiple subtypes (14, 17, 31). Subtypes A, B, and C in India have been reported (4, 22, 30, 31, 59), but mosaic HIV-1 there has not been reported. The existence of such recombinants makes characterization of variants by analyzing subgenomic segments incomplete. Second, immune responses to vaccines based on single genes such as env have been limited (13), and attention is being shifted toward multivalent vaccines that incorporate other gene products. Third, interactions among discontinuous regions of the genome, such as between the long terminal repeat (LTR) and pol (26), can be detected only when such regions can be analyzed from the same template. In an effort to characterize subtype C virus genomes being transmitted currently in India, viral isolates were obtained from individuals with seroincident infections in India. Three of the isolates (collected in 1994 and 1995) were known to be non-syncytium inducing (NSI) and therefore resembled viruses transmitted through unprotected sexual contact, which account for 75 to 85% of new infections (2, 15, 61). These isolates were cloned, and nearly full-length genomic sequences were determined. Detailed sequence analysis was performed, as was an analysis of variation in characterized cytotoxic T lymphocyte (CTL) epitopes. read more

Topics:

Population (53%)53% related to the paper

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2,472 Citations

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Journal of Virology format uses unsrt asm custom citation citation style.

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Frequently asked questions

1. Can I write Journal of Virology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Journal of Virology guidelines and auto format it.

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Yes, the template is compliant with the Journal of Virology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Journal of Virology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Journal of Virology citation style.

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Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Journal of Virology.

5. Can I use a manuscript in Journal of Virology that I have written in MS Word?

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7. Where can I find the template for the Journal of Virology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Journal of Virology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Journal of Virology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Journal of Virology an online tool or is there a desktop version?

SciSpace's Journal of Virology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

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11. What is the output that I would get after using Journal of Virology?

After writing your paper autoformatting in Journal of Virology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Journal of Virology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Journal of Virology?

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Journal of Virology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour	Archiving policy
Green	Can archive pre-print and post-print or publisher's version/PDF
Blue	Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow	Can archive pre-print (ie pre-refereeing)
White	Archiving not formally supported

FYI:

Pre-prints as being the version of the paper before peer review and
Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Journal of Virology?

The 5 most common citation types in order of usage for Journal of Virology are:.

S. No.	Citation Style Type
1.	Author Year
2.	Numbered
3.	Numbered (Superscripted)
4.	Author Year (Cited Pages)
5.	Footnote

15. How do I submit my article to the Journal of Virology?

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Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Journal of Virology Endnote style according to Elsevier guidelines.

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Journal of Virology — Template for authors

Related Journals

Journal Performance & Insights

Impact Factor

CiteRatio

4.501

8.9

Insights

Insights

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

2.617

1.372

Insights

Insights

Journal of Virology

Journal of Virology

Top papers written in this journal

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What to expect from SciSpace?

Speed and accuracy over MS Word

Plagiarism Reports via Turnitin

Freedom from formatting guidelines

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Frequently asked questions

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No word template required

Verifed journal formats

Trusted by academicians

Fast and reliable,
built for complaince.