Example of Nature Cell Biology format
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Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format
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Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format Example of Nature Cell Biology format
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open access Open Access
recommended Recommended

Nature Cell Biology — Template for authors

Publisher: Nature
Categories Rank Trend in last 3 yrs
Cell Biology #6 of 279 up up by 2 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 576 Published Papers | 18166 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 28/06/2020
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Related Journals

open access Open Access
recommended Recommended

Taylor and Francis

Quality:  
High
CiteRatio: 15.1
SJR: 3.934
SNIP: 2.351
open access Open Access

American Chemical Society

Quality:  
High
CiteRatio: 6.5
SJR: 1.158
SNIP: 1.002
open access Open Access

Nature

Quality:  
High
CiteRatio: 6.7
SJR: 1.542
SNIP: 1.216
open access Open Access
recommended Recommended

Nature

Quality:  
High
CiteRatio: 28.2
SJR: 7.305
SNIP: 3.41

Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

20.042

13% from 2018

Impact factor for Nature Cell Biology from 2016 - 2019
Year Value
2019 20.042
2018 17.728
2017 19.064
2016 20.06
graph view Graph view
table view Table view

31.5

25% from 2019

CiteRatio for Nature Cell Biology from 2016 - 2020
Year Value
2020 31.5
2019 25.3
2018 28.2
2017 29.4
2016 31.7
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 13% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 25% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

11.38

15% from 2019

SJR for Nature Cell Biology from 2016 - 2020
Year Value
2020 11.38
2019 9.916
2018 12.137
2017 14.07
2016 14.696
graph view Graph view
table view Table view

3.716

24% from 2019

SNIP for Nature Cell Biology from 2016 - 2020
Year Value
2020 3.716
2019 3.005
2018 3.284
2017 3.557
2016 3.602
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 15% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 24% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Nature Cell Biology

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Nature

Nature Cell Biology

Nature Cell Biology publishes papers of the highest quality from all areas of cell biology, encouraging those that shed light on the molecular mechanisms responsible for fundamental cell biological processes. The journal's scope is broad and includes the following areas (not l...... Read More

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Last updated on
28 Jun 2020
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ISSN
1476-4679
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Acceptance Rate
Not provided
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Frequency
Not provided
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Open Access
Not provided
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Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Naturemag Citation
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Citation Type
Numbered (Superscripted)
25
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Bibliography Example
Beenakker, C. W. J. Specular andreev reflection in graphene. Phys. Rev. Lett. 97, 067007 (2006). URL 10.1103/PhysRevLett.97.067007.

Top papers written in this journal

Journal Article DOI: 10.1038/NCB1596
Exosome-mediated transfer of mRNAs and microRNAs is a novel mechanism of genetic exchange between cells
Hadi Valadi1, Karin M. Ekström1, Apostolos Bossios1, Margareta Sjöstrand1, James J. Lee2, Jan Lötvall1
07 May 2007 - Nature Cell Biology

Abstract:

Exosomes are vesicles of endocytic origin released by many cells. These vesicles can mediate communication between cells, facilitating processes such as antigen presentation. Here, we show that exosomes from a mouse and a human mast cell line (MC/9 and HMC-1, respectively), as well as primary bone marrow-derived mouse mast ce... Exosomes are vesicles of endocytic origin released by many cells. These vesicles can mediate communication between cells, facilitating processes such as antigen presentation. Here, we show that exosomes from a mouse and a human mast cell line (MC/9 and HMC-1, respectively), as well as primary bone marrow-derived mouse mast cells, contain RNA. Microarray assessments revealed the presence of mRNA from approximately 1300 genes, many of which are not present in the cytoplasm of the donor cell. In vitro translation proved that the exosome mRNAs were functional. Quality control RNA analysis of total RNA derived from exosomes also revealed presence of small RNAs, including microRNAs. The RNA from mast cell exosomes is transferable to other mouse and human mast cells. After transfer of mouse exosomal RNA to human mast cells, new mouse proteins were found in the recipient cells, indicating that transferred exosomal mRNA can be translated after entering another cell. In summary, we show that exosomes contain both mRNA and microRNA, which can be delivered to another cell, and can be functional in this new location. We propose that this RNA is called "exosomal shuttle RNA" (esRNA). read more read less

Topics:

Exosome (69%)69% related to the paper, RNA silencing (61%)61% related to the paper, RNA (60%)60% related to the paper, Extracellular RNA (60%)60% related to the paper, Exosomal secretion (58%)58% related to the paper
View PDF
10,484 Citations
open accessOpen access Journal Article DOI: 10.1038/NCB2152
AMPK and mTOR regulate autophagy through direct phosphorylation of Ulk1
Joungmok Kim1, Mondira Kundu2, Benoit Viollet3, Kun-Liang Guan1
01 Feb 2011 - Nature Cell Biology

Abstract:

Autophagy is a process by which components of the cell are degraded to maintain essential activity and viability in response to nutrient limitation. Extensive genetic studies have shown that the yeast ATG1 kinase has an essential role in autophagy induction. Furthermore, autophagy is promoted by AMP activated protein kinase (... Autophagy is a process by which components of the cell are degraded to maintain essential activity and viability in response to nutrient limitation. Extensive genetic studies have shown that the yeast ATG1 kinase has an essential role in autophagy induction. Furthermore, autophagy is promoted by AMP activated protein kinase (AMPK), which is a key energy sensor and regulates cellular metabolism to maintain energy homeostasis. Conversely, autophagy is inhibited by the mammalian target of rapamycin (mTOR), a central cell-growth regulator that integrates growth factor and nutrient signals. Here we demonstrate a molecular mechanism for regulation of the mammalian autophagy-initiating kinase Ulk1, a homologue of yeast ATG1. Under glucose starvation, AMPK promotes autophagy by directly activating Ulk1 through phosphorylation of Ser 317 and Ser 777. Under nutrient sufficiency, high mTOR activity prevents Ulk1 activation by phosphorylating Ulk1 Ser 757 and disrupting the interaction between Ulk1 and AMPK. This coordinated phosphorylation is important for Ulk1 in autophagy induction. Our study has revealed a signalling mechanism for Ulk1 regulation and autophagy induction in response to nutrient signalling. read more read less

Topics:

Atg1 (64%)64% related to the paper, Autophagy database (64%)64% related to the paper, Autophagy-Related Protein-1 Homolog (63%)63% related to the paper, AMPK (62%)62% related to the paper, Autophagy (61%)61% related to the paper
View PDF
5,314 Citations
open accessOpen access Journal Article DOI: 10.1038/NCB1800
Glioblastoma microvesicles transport RNA and proteins that promote tumour growth and provide diagnostic biomarkers
01 Dec 2008 - Nature Cell Biology

Abstract:

Glioblastoma tumour cells release microvesicles (exosomes) containing mRNA, miRNA and angiogenic proteins. These microvesicles are taken up by normal host cells, such as brain microvascular endothelial cells. By incorporating an mRNA for a reporter protein into these microvesicles, we demonstrate that messages delivered by mi... Glioblastoma tumour cells release microvesicles (exosomes) containing mRNA, miRNA and angiogenic proteins. These microvesicles are taken up by normal host cells, such as brain microvascular endothelial cells. By incorporating an mRNA for a reporter protein into these microvesicles, we demonstrate that messages delivered by microvesicles are translated by recipient cells. These microvesicles are also enriched in angiogenic proteins and stimulate tubule formation by endothelial cells. Tumour-derived microvesicles therefore serve as a means of delivering genetic information and proteins to recipient cells in the tumour environment. Glioblastoma microvesicles also stimulated proliferation of a human glioma cell line, indicating a self-promoting aspect. Messenger RNA mutant/variants and miRNAs characteristic of gliomas could be detected in serum microvesicles of glioblastoma patients. The tumour-specific EGFRvIII was detected in serum microvesicles from 7 out of 25 glioblastoma patients. Thus, tumour-derived microvesicles may provide diagnostic information and aid in therapeutic decisions for cancer patients through a blood test. read more read less

Topics:

Microvesicles (72%)72% related to the paper, Circulating microvesicle (64%)64% related to the paper, Microvesicle (58%)58% related to the paper, Extracellular RNA (54%)54% related to the paper
View PDF
4,118 Citations
Journal Article DOI: 10.1038/NCB1722
The miR-200 family and miR-205 regulate epithelial to mesenchymal transition by targeting ZEB1 and SIP1.
01 May 2008 - Nature Cell Biology

Abstract:

Epithelial to mesenchymal transition (EMT) facilitates tissue remodelling during embryonic development and is viewed as an essential early step in tumour metastasis. We found that all five members of the microRNA-200 family (miR-200a, miR-200b, miR-200c, miR-141 and miR-429) and miR-205 were markedly downregulated in cells th... Epithelial to mesenchymal transition (EMT) facilitates tissue remodelling during embryonic development and is viewed as an essential early step in tumour metastasis. We found that all five members of the microRNA-200 family (miR-200a, miR-200b, miR-200c, miR-141 and miR-429) and miR-205 were markedly downregulated in cells that had undergone EMT in response to transforming growth factor (TGF)-β or to ectopic expression of the protein tyrosine phosphatase Pez. Enforced expression of the miR-200 family alone was sufficient to prevent TGF-β-induced EMT. Together, these microRNAs cooperatively regulate expression of the E-cadherin transcriptional repressors ZEB1 (also known as δEF1) and SIP1 (also known as ZEB2), factors previously implicated in EMT and tumour metastasis. Inhibition of the microRNAs was sufficient to induce EMT in a process requiring upregulation of ZEB1 and/or SIP1. Conversely, ectopic expression of these microRNAs in mesenchymal cells initiated mesenchymal to epithelial transition (MET). Consistent with their role in regulating EMT, expression of these microRNAs was found to be lost in invasive breast cancer cell lines with mesenchymal phenotype. Expression of the miR-200 family was also lost in regions of metaplastic breast cancer specimens lacking E-cadherin. These data suggest that downregulation of the microRNAs may be an important step in tumour progression. MicroRNAs are small, non-coding RNAs that modulate gene expression post-transcriptionally. In metazoa, they act predominantly to inhibit translation of their specific targets, but they also typically cause a modest reduction in the level of their target mRNAs 1,2 . Hundreds of microRNAs have been identified in vertebrates, with varying patterns of expression that range from ubiquitous to highly tissue- or developmental-stage-restricted. In some cases, an individual microRNA can act as a developmental switch by regulating a key target mRNA 3 . Speculating that switching between cell phenotypes that occurs during EMT may be specified to some extent by microRNAs, we searched for microRNAs whose expression changed during EMT. To this end, we used an in vitro model of EMT, which was generated by stable transfection of Madin Darby canine kidney (MDCK) epithelial cells with the protein tyrosine phosphatase Pez (PTP-Pez). Overexpression of PTP-Pez caused MDCK cells to undergo EMT, as indicated by loss of E-cadherin expression, gain in expression of the mesenchymal markers fibronectin, ZEB1 and SIP1, loss of cohesion, induction of cell motility and a change in cell morphology read more read less

Topics:

Epithelial–mesenchymal transition (60%)60% related to the paper, Ectopic expression (57%)57% related to the paper, microRNA (53%)53% related to the paper, Signal transduction (51%)51% related to the paper, Cell cycle (51%)51% related to the paper
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3,640 Citations
Journal Article DOI: 10.1038/35000025
The transcription factor Snail controls epithelial–mesenchymal transitions by repressing E-cadherin expression
01 Feb 2000 - Nature Cell Biology

Abstract:

The Snail family of transcription factors has previously been implicated in the differentiation of epithelial cells into mesenchymal cells (epithelial-mesenchymal transitions) during embryonic development. Epithelial-mesenchymal transitions are also determinants of the progression of carcinomas, occurring concomitantly with t... The Snail family of transcription factors has previously been implicated in the differentiation of epithelial cells into mesenchymal cells (epithelial-mesenchymal transitions) during embryonic development. Epithelial-mesenchymal transitions are also determinants of the progression of carcinomas, occurring concomitantly with the cellular acquisition of migratory properties following downregulation of expression of the adhesion protein E-cadherin. Here we show that mouse Snail is a strong repressor of transcription of the E-cadherin gene. Epithelial cells that ectopically express Snail adopt a fibroblastoid phenotype and acquire tumorigenic and invasive properties. Endogenous Snail protein is present in invasive mouse and human carcinoma cell lines and tumours in which E-cadherin expression has been lost. Therefore, the same molecules are used to trigger epithelial-mesenchymal transitions during embryonic development and in tumour progression. Snail may thus be considered as a marker for malignancy, opening up new avenues for the design of specific anti-invasive drugs. read more read less

Topics:

SNAI1 (67%)67% related to the paper, Snail (58%)58% related to the paper, Mesenchymal–epithelial transition (57%)57% related to the paper, Cellular differentiation (55%)55% related to the paper, Transcription factor (53%)53% related to the paper
3,426 Citations
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Nature Cell Biology format uses Naturemag Citation citation style.

Automatically format and order your citations and bibliography in a click.

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Frequently asked questions

1. Can I write Nature Cell Biology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Nature Cell Biology guidelines and auto format it.

2. Do you follow the Nature Cell Biology guidelines?

Yes, the template is compliant with the Nature Cell Biology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Nature Cell Biology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Nature Cell Biology citation style.

4. Can I use the Nature Cell Biology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Nature Cell Biology.

5. Can I use a manuscript in Nature Cell Biology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Nature Cell Biology that you can download at the end.

6. How long does it usually take you to format my papers in Nature Cell Biology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Nature Cell Biology.

7. Where can I find the template for the Nature Cell Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Nature Cell Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Nature Cell Biology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Nature Cell Biology an online tool or is there a desktop version?

SciSpace's Nature Cell Biology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Nature Cell Biology?

Sure. You can request any template and we'll have it setup within a few days. You can find the request box in Journal Gallery on the right side bar under the heading, "Couldn't find the format you were looking for like Nature Cell Biology?”

11. What is the output that I would get after using Nature Cell Biology?

After writing your paper autoformatting in Nature Cell Biology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Nature Cell Biology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Nature Cell Biology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Nature Cell Biology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Nature Cell Biology?

The 5 most common citation types in order of usage for Nature Cell Biology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Nature Cell Biology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Nature Cell Biology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Nature Cell Biology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Nature Cell Biology Endnote style according to Elsevier guidelines.

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