Example of Seminars in Immunopathology format
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Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format
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Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format Example of Seminars in Immunopathology format
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Seminars in Immunopathology — Template for authors

Publisher: Springer
Categories Rank Trend in last 3 yrs
Immunology and Allergy #9 of 182 up up by 11 ranks
Immunology #10 of 202 up up by 14 ranks
journal-quality-icon Journal quality:
High
calendar-icon Last 4 years overview: 203 Published Papers | 3882 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 27/06/2020
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Related Journals

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Journal Performance & Insights

Impact Factor

CiteRatio

Determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

A measure of average citations received per peer-reviewed paper published in the journal.

7.48

10% from 2018

Impact factor for Seminars in Immunopathology from 2016 - 2019
Year Value
2019 7.48
2018 6.804
2017 6.437
2016 5.296
graph view Graph view
table view Table view

19.1

40% from 2019

CiteRatio for Seminars in Immunopathology from 2016 - 2020
Year Value
2020 19.1
2019 13.6
2018 12.5
2017 10.3
2016 11.5
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has increased by 10% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

insights Insights

  • CiteRatio of this journal has increased by 40% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR)

Source Normalized Impact per Paper (SNIP)

Measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

Measures actual citations received relative to citations expected for the journal's category.

4.778

64% from 2019

SJR for Seminars in Immunopathology from 2016 - 2020
Year Value
2020 4.778
2019 2.92
2018 2.691
2017 2.874
2016 3.277
graph view Graph view
table view Table view

5.563

192% from 2019

SNIP for Seminars in Immunopathology from 2016 - 2020
Year Value
2020 5.563
2019 1.903
2018 1.772
2017 1.576
2016 1.601
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has increased by 64% in last years.
  • This journal’s SJR is in the top 10 percentile category.

insights Insights

  • SNIP of this journal has increased by 192% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Seminars in Immunopathology

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Springer

Seminars in Immunopathology

The aim of this journal is to bring clinicians and pathologists up-to-date on developments in the field of immunopathology. For this purpose topical issues will be organized usually with the help of a guest editor. Recent developments are summarized in review articles by autho...... Read More

Immunology and Allergy

Medicine

i
Last updated on
27 Jun 2020
i
ISSN
1863-2297
i
Impact Factor
High - 1.575
i
Open Access
No
i
Sherpa RoMEO Archiving Policy
Green faq
i
Plagiarism Check
Available via Turnitin
i
Endnote Style
Download Available
i
Bibliography Name
SPBASIC
i
Citation Type
Author Year
(Blonder et al, 1982)
i
Bibliography Example
Beenakker CWJ (2006) Specular andreev reflection in graphene. Phys Rev Lett 97(6):067,007, URL 10.1103/PhysRevLett.97.067007

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.1007/S00281-017-0629-X
Pathogenic human coronavirus infections: causes and consequences of cytokine storm and immunopathology
Rudragouda Channappanavar1, Stanley Perlman1

Abstract:

Human coronaviruses (hCoVs) can be divided into low pathogenic and highly pathogenic coronaviruses. The low pathogenic CoVs infect the upper respiratory tract and cause mild, cold-like respiratory illness. In contrast, highly pathogenic hCoVs such as severe acute respiratory syndrome CoV (SARS-CoV) and Middle East respiratory... Human coronaviruses (hCoVs) can be divided into low pathogenic and highly pathogenic coronaviruses. The low pathogenic CoVs infect the upper respiratory tract and cause mild, cold-like respiratory illness. In contrast, highly pathogenic hCoVs such as severe acute respiratory syndrome CoV (SARS-CoV) and Middle East respiratory syndrome CoV (MERS-CoV) predominantly infect lower airways and cause fatal pneumonia. Severe pneumonia caused by pathogenic hCoVs is often associated with rapid virus replication, massive inflammatory cell infiltration and elevated pro-inflammatory cytokine/chemokine responses resulting in acute lung injury (ALI), and acute respiratory distress syndrome (ARDS). Recent studies in experimentally infected animal strongly suggest a crucial role for virus-induced immunopathological events in causing fatal pneumonia after hCoV infections. Here we review the current understanding of how a dysregulated immune response may cause lung immunopathology leading to deleterious clinical manifestations after pathogenic hCoV infections. read more read less

Topics:

Middle East respiratory syndrome (57%)57% related to the paper, Pneumonia (55%)55% related to the paper, ARDS (55%)55% related to the paper, Cytokine storm (55%)55% related to the paper, Lung injury (54%)54% related to the paper
View PDF
1,984 Citations
Journal Article DOI: 10.1007/S00281-017-0639-8
Cytokine storm and sepsis disease pathogenesis
Benjamin G. Chousterman, Filip K. Swirski1, Georg F. Weber2

Abstract:

Infectious diseases are a leading cause of death worldwide. Sepsis is a severe clinical syndrome related to the host response to infection. The severity of infections is due to an activation cascade that will lead to an autoamplifying cytokine production: the cytokine storm. Cytokines are a broad category of relatively small ... Infectious diseases are a leading cause of death worldwide. Sepsis is a severe clinical syndrome related to the host response to infection. The severity of infections is due to an activation cascade that will lead to an autoamplifying cytokine production: the cytokine storm. Cytokines are a broad category of relatively small proteins (<40 kDa) that are produced and released with the aim of cell signaling. Our understanding of the processes that trigger this tremendous amount of cytokine production has made dramatic progress over the last decades, but unfortunately, these findings could not translate yet into effective treatments; so far, all clinical trials targeting cytokine production or effects failed. This review aims to summarize the pathophysiology of the cytokine storm; to describe the type, effects, and kinetics of cytokine production; and to discuss the therapeutic challenges of targeting cytokines. New promising therapeutic strategies focusing on the endothelium, as a source and a target of cytokines, are described. read more read less

Topics:

Cytokine storm (66%)66% related to the paper, Cytokine (55%)55% related to the paper
763 Citations
Journal Article DOI: 10.1007/S00281-011-0290-8
Fibrinogen as a key regulator of inflammation in disease
Dimitrios Davalos1, Katerina Akassoglou1

Abstract:

The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles in the acute hemostatic cascade. Key molecular players of the coagulation cascade like tissue factor, thrombin, and fibrinogen are epidemiologically and mechanistically... The interaction of coagulation factors with the perivascular environment affects the development of disease in ways that extend beyond their traditional roles in the acute hemostatic cascade. Key molecular players of the coagulation cascade like tissue factor, thrombin, and fibrinogen are epidemiologically and mechanistically linked with diseases with an inflammatory component. Moreover, the identification of novel molecular mechanisms linking coagulation and inflammation has highlighted factors of the coagulation cascade as new targets for therapeutic intervention in a wide range of inflammatory human diseases. In particular, a proinflammatory role for fibrinogen has been reported in vascular wall disease, stroke, spinal cord injury, brain trauma, multiple sclerosis, Alzheimer’s disease, rheumatoid arthritis, bacterial infection, colitis, lung and kidney fibrosis, Duchenne muscular dystrophy, and several types of cancer. Genetic and pharmacologic studies have unraveled pivotal roles for fibrinogen in determining the extent of local or systemic inflammation. As cellular and molecular mechanisms for fibrinogen functions in tissues are identified, the role of fibrinogen is evolving from a marker of vascular rapture to a multi-faceted signaling molecule with a wide spectrum of functions that can tip the balance between hemostasis and thrombosis, coagulation and fibrosis, protection from infection and extensive inflammation, and eventually life and death. This review will discuss some of the main molecular links between coagulation and inflammation and will focus on the role of fibrinogen in inflammatory disease highlighting its unique structural properties, cellular targets, and signal transduction pathways that make it a potent proinflammatory mediator and a potential therapeutic target. read more read less

Topics:

Fibrinogen (57%)57% related to the paper, Proinflammatory cytokine (54%)54% related to the paper, Thrombin (52%)52% related to the paper, Inflammation (52%)52% related to the paper, Coagulation (51%)51% related to the paper
713 Citations
open accessOpen access Journal Article DOI: 10.1007/S00281-009-0177-0
The blood-brain and the blood-cerebrospinal fluid barriers: function and dysfunction
Britta Engelhardt1, Lydia Sorokin2

Abstract:

The central nervous system (CNS) is tightly sealed from the changeable milieu of blood by the blood-brain barrier (BBB) and the blood-cerebrospinal fluid (CSF) barrier (BCSFB). While the BBB is considered to be localized at the level of the endothelial cells within CNS microvessels, the BCSFB is established by choroid plexus ... The central nervous system (CNS) is tightly sealed from the changeable milieu of blood by the blood-brain barrier (BBB) and the blood-cerebrospinal fluid (CSF) barrier (BCSFB). While the BBB is considered to be localized at the level of the endothelial cells within CNS microvessels, the BCSFB is established by choroid plexus epithelial cells. The BBB inhibits the free paracellular diffusion of water-soluble molecules by an elaborate network of complex tight junctions (TJs) that interconnects the endothelial cells. Combined with the absence of fenestrae and an extremely low pinocytotic activity, which inhibit transcellular passage of molecules across the barrier, these morphological peculiarities establish the physical permeability barrier of the BBB. In addition, a functional BBB is manifested by a number of permanently active transport mechanisms, specifically expressed by brain capillary endothelial cells that ensure the transport of nutrients into the CNS and exclusion of blood-borne molecules that could be detrimental to the milieu required for neural transmission. Finally, while the endothelial cells constitute the physical and metabolic barrier per se, interactions with adjacent cellular and acellular layers are prerequisites for barrier function. The fully differentiated BBB consists of a complex system comprising the highly specialized endothelial cells and their underlying basement membrane in which a large number of pericytes are embedded, perivascular antigen-presenting cells, and an ensheathment of astrocytic endfeet and associated parenchymal basement membrane. Endothelial cell morphology, biochemistry, and function thus make these brain microvascular endothelial cells unique and distinguishable from all other endothelial cells in the body. Similar to the endothelial barrier, the morphological correlate of the BCSFB is found at the level of unique apical tight junctions between the choroid plexus epithelial cells inhibiting paracellular diffusion of water-soluble molecules across this barrier. Besides its barrier function, choroid plexus epithelial cells have a secretory function and produce the CSF. The barrier and secretory function of the choroid plexus epithelial cells are maintained by the expression of numerous transport systems allowing the directed transport of ions and nutrients into the CSF and the removal of toxic agents out of the CSF. In the event of CNS pathology, barrier characteristics of the blood-CNS barriers are altered, leading to edema formation and recruitment of inflammatory cells into the CNS. In this review we will describe current knowledge on the cellular and molecular basis of the functional and dysfunctional blood-CNS barriers with focus on CNS autoimmune inflammation. read more read less

Topics:

Blood–brain barrier (62%)62% related to the paper, Choroid plexus (61%)61% related to the paper, Drug delivery to the brain (60%)60% related to the paper, Neuroinflammation (58%)58% related to the paper, Barrier function (57%)57% related to the paper
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631 Citations
Journal Article DOI: 10.1007/S00281-011-0247-Y
Prognostic significance of autoimmunity during treatment of melanoma with interferon
Michal T. Krauze1, Ahmad A. Tarhini1, Helen Gogas2, John M. Kirkwood1

Abstract:

Since the pivotal cooperative group trials in the 1980’s-90’s,, high-dose interferon (HDI) has been the standard of adjuvant therapy. Despite multiple other trials evaluating potential new therapies in melanoma, HDI remains the only FDA-approved therapy for stage IIB and III melanoma. Initial reports from the more recent phas... Since the pivotal cooperative group trials in the 1980’s-90’s,, high-dose interferon (HDI) has been the standard of adjuvant therapy. Despite multiple other trials evaluating potential new therapies in melanoma, HDI remains the only FDA-approved therapy for stage IIB and III melanoma. Initial reports from the more recent phase III international trials of modifications of the original HDI regimen linked the appearance of autoimmunity with improved outcomes of disease. Trials of high-dose interleukin-2, many years earlier, reported anecdotal observations that were consistent with the hypothesis that autoimmunity and clinical benefit of immunotherapies of melanoma are linked with one another. The only prospectively conducted study examining the appearance of clinical and laboratory evidence of autoimmunity during HDI therapy was published by Gogas and colleagues, demonstrating statistically significant impact on relapse-free survival and overall survival. Retrospectively conducted studies of different intermediate dosage regimens of interferon (IFN) have not fully confirmed the linkage of serological evidence of autoimmunity and improved survival outcomes. With the emergence of new immunotherapies in treatment of melanoma, this review highlights the importance of autoimmunity for future applications in melanoma and reviews significant differences of past studies evaluating the appearance of autoimmunity during IFN therapy in high-risk melanoma. read more read less

Topics:

Adjuvant therapy (52%)52% related to the paper
View PDF
588 Citations
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Frequently asked questions

1. Can I write Seminars in Immunopathology in LaTeX?

Absolutely not! Our tool has been designed to help you focus on writing. You can write your entire paper as per the Seminars in Immunopathology guidelines and auto format it.

2. Do you follow the Seminars in Immunopathology guidelines?

Yes, the template is compliant with the Seminars in Immunopathology guidelines. Our experts at SciSpace ensure that. If there are any changes to the journal's guidelines, we'll change our algorithm accordingly.

3. Can I cite my article in multiple styles in Seminars in Immunopathology?

Of course! We support all the top citation styles, such as APA style, MLA style, Vancouver style, Harvard style, and Chicago style. For example, when you write your paper and hit autoformat, our system will automatically update your article as per the Seminars in Immunopathology citation style.

4. Can I use the Seminars in Immunopathology templates for free?

Sign up for our free trial, and you'll be able to use all our features for seven days. You'll see how helpful they are and how inexpensive they are compared to other options, Especially for Seminars in Immunopathology.

5. Can I use a manuscript in Seminars in Immunopathology that I have written in MS Word?

Yes. You can choose the right template, copy-paste the contents from the word document, and click on auto-format. Once you're done, you'll have a publish-ready paper Seminars in Immunopathology that you can download at the end.

6. How long does it usually take you to format my papers in Seminars in Immunopathology?

It only takes a matter of seconds to edit your manuscript. Besides that, our intuitive editor saves you from writing and formatting it in Seminars in Immunopathology.

7. Where can I find the template for the Seminars in Immunopathology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Seminars in Immunopathology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

8. Can I reformat my paper to fit the Seminars in Immunopathology's guidelines?

Of course! You can do this using our intuitive editor. It's very easy. If you need help, our support team is always ready to assist you.

9. Seminars in Immunopathology an online tool or is there a desktop version?

SciSpace's Seminars in Immunopathology is currently available as an online tool. We're developing a desktop version, too. You can request (or upvote) any features that you think would be helpful for you and other researchers in the "feature request" section of your account once you've signed up with us.

10. I cannot find my template in your gallery. Can you create it for me like Seminars in Immunopathology?

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After writing your paper autoformatting in Seminars in Immunopathology, you can download it in multiple formats, viz., PDF, Docx, and LaTeX.

12. Is Seminars in Immunopathology's impact factor high enough that I should try publishing my article there?

To be honest, the answer is no. The impact factor is one of the many elements that determine the quality of a journal. Few of these factors include review board, rejection rates, frequency of inclusion in indexes, and Eigenfactor. You need to assess all these factors before you make your final call.

13. What is Sherpa RoMEO Archiving Policy for Seminars in Immunopathology?

SHERPA/RoMEO Database

We extracted this data from Sherpa Romeo to help researchers understand the access level of this journal in accordance with the Sherpa Romeo Archiving Policy for Seminars in Immunopathology. The table below indicates the level of access a journal has as per Sherpa Romeo's archiving policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

14. What are the most common citation types In Seminars in Immunopathology?

The 5 most common citation types in order of usage for Seminars in Immunopathology are:.

S. No. Citation Style Type
1. Author Year
2. Numbered
3. Numbered (Superscripted)
4. Author Year (Cited Pages)
5. Footnote

15. How do I submit my article to the Seminars in Immunopathology?

It is possible to find the Word template for any journal on Google. However, why use a template when you can write your entire manuscript on SciSpace , auto format it as per Seminars in Immunopathology's guidelines and download the same in Word, PDF and LaTeX formats? Give us a try!.

16. Can I download Seminars in Immunopathology in Endnote format?

Yes, SciSpace provides this functionality. After signing up, you would need to import your existing references from Word or Bib file to SciSpace. Then SciSpace would allow you to download your references in Seminars in Immunopathology Endnote style according to Elsevier guidelines.

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