Example of Current HIV Research format
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Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format
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Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format Example of Current HIV Research format
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open access Open Access ISSN: 1570162X e-ISSN: 18734251

Current HIV Research — Template for authors

Publisher: Bentham Science
Categories Rank Trend in last 3 yrs
Infectious Diseases #202 of 288 down down by 64 ranks
Virology #55 of 69 down down by 8 ranks
journal-quality-icon Journal quality:
Medium
calendar-icon Last 4 years overview: 193 Published Papers | 334 Citations
indexed-in-icon Indexed in: Scopus
last-updated-icon Last updated: 17/06/2020
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FAQ

Journal Performance & Insights

  • Impact Factor
  • CiteRatio
  • SJR
  • SNIP

Impact factor determines the importance of a journal by taking a measure of frequency with which the average article in a journal has been cited in a particular year.

0.802

28% from 2018

Impact factor for Current HIV Research from 2016 - 2019
Year Value
2019 0.802
2018 1.115
2017 1.562
2016 1.612
graph view Graph view
table view Table view

insights Insights

  • Impact factor of this journal has decreased by 28% in last year.
  • This journal’s impact factor is in the top 10 percentile category.

CiteRatio is a measure of average citations received per peer-reviewed paper published in the journal.

1.7

26% from 2019

CiteRatio for Current HIV Research from 2016 - 2020
Year Value
2020 1.7
2019 2.3
2018 2.4
2017 2.7
2016 2.9
graph view Graph view
table view Table view

insights Insights

  • CiteRatio of this journal has decreased by 26% in last years.
  • This journal’s CiteRatio is in the top 10 percentile category.

SCImago Journal Rank (SJR) measures weighted citations received by the journal. Citation weighting depends on the categories and prestige of the citing journal.

0.561

5% from 2019

SJR for Current HIV Research from 2016 - 2020
Year Value
2020 0.561
2019 0.588
2018 0.632
2017 0.845
2016 0.931
graph view Graph view
table view Table view

insights Insights

  • SJR of this journal has decreased by 5% in last years.
  • This journal’s SJR is in the top 10 percentile category.

Source Normalized Impact per Paper (SNIP) measures actual citations received relative to citations expected for the journal's category.

0.418

10% from 2019

SNIP for Current HIV Research from 2016 - 2020
Year Value
2020 0.418
2019 0.38
2018 0.441
2017 0.46
2016 0.568
graph view Graph view
table view Table view

insights Insights

  • SNIP of this journal has increased by 10% in last years.
  • This journal’s SNIP is in the top 10 percentile category.

Related Journals

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CiteRatio: 6.0 | SJR: 2.135 | SNIP: 1.267
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Springer

CiteRatio: 6.0 | SJR: 1.026 | SNIP: 1.34
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Elsevier

CiteRatio: 5.9 | SJR: 1.175 | SNIP: 1.033
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CiteRatio: 23.9 | SJR: 4.491 | SNIP: 3.727
Current HIV Research

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Bentham Science

Current HIV Research

Current HIV Research aims to cover all the latest and outstanding developments of HIV research. The journal publishes comprehensive review articles, letters, and novel, pioneering work in the basic and clinical fields on all areas of HIV research, covering virus replication an...... Read More

Infectious Diseases

Virology

Medicine

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Last updated on
16 Jun 2020
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ISSN
1570-162X
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Impact Factor
Medium - 0.629
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Open Access
No
i
Sherpa RoMEO Archiving Policy
Yellow faq
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Plagiarism Check
Available via Turnitin
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Endnote Style
Download Available
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Bibliography Name
Vancouver
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Citation Type
Numbered
[25]
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Bibliography Example
Blonder, G E, Tinkham, M, & Klapwijk, T M. Transition from metallic to tunnel- ing regimes in superconducting microconstrictions: Excess current, charge imbalance, and supercurrent conversion. Phys. Rev. B. 2013;87(10):100510.

Top papers written in this journal

open accessOpen access Journal Article DOI: 10.2174/157016208785861195
Cellular Reservoirs of HIV-1 and their Role in Viral Persistence
Aikaterini Alexaki1, Yujie Liu, Brian Wigdahl1
31 Aug 2008 - Current HIV Research

Abstract:

A major obstacle in human immunodeficiency virus type 1 (HIV-1) eradication is the ability of the virus to remain latent in a subpopulation of the cells it infects. Latently infected cells can escape the viral immune response and persist for long periods of time, despite the presence of successful highly active antiretroviral... A major obstacle in human immunodeficiency virus type 1 (HIV-1) eradication is the ability of the virus to remain latent in a subpopulation of the cells it infects. Latently infected cells can escape the viral immune response and persist for long periods of time, despite the presence of successful highly active antiretroviral therapy (HAART). Given the appropriate stimulus, latently infected cells can reactivate and start producing infectious virions. The susceptibility of these cell populations to HIV-1, their life span, their proliferative capacity, and their ability to periodically produce infectious virus subsequent to alterations in cellular physiology and/or immunologic controls are critical issues which determine the contribution of these cells to viral persistence. Memory CD4+ T cells due to the long life span, which may be several years, and their ability to reactivate upon encounter with their cognate antigen or other stimulation, are considered a critical reservoir for maintenance of latent HIV-1 proviral DNA. Cells of the monocyte-macrophage lineage, which originate in the bone marrow (BM), are of particular importance in HIV-1 persistence due to their ability to cross the blood-brain barrier (BBB) and spread HIV-1 infection in the immunoprivileged central nervous system (CNS). Hematopoietic progenitor cells (HPCs) are also a potential HIV-1 reservoir, as several studies have shown that CD34+ HPCs carrying proviral DNA can be found in vivo in a subpopulation of HIV-1-infected patients. The ability of HPCs to proliferate and potentially generate clonal populations of infected cells of the monocyte-macrophage lineage may be crucial in HIV-1 dissemination. The contribution of these and other cell populations in HIV-1 persistence, as well as the possible strategies to eliminate latently infected cells are critically examined in this review. read more read less

Topics:

Virus latency (57%)57% related to the paper, Immune system (51%)51% related to the paper, Virus (51%)51% related to the paper, Antigen (50%)50% related to the paper
312 Citations
Journal Article DOI: 10.2174/157016207781023947
The Antimicrobial Peptide LL-37 Inhibits HIV-1 Replication
30 Jun 2007 - Current HIV Research

Abstract:

The antimicrobial peptide LL-37 is the only cathelicidin that has been described in humans. LL-37 exerts chemotactic, immunomodulatory and angiogenic effects; activities that are mediated through binding to the formyl peptide receptor like (FPRL)-1 receptor. Agonistic ligation of FPRL-1 can also induce down-regulation of HIV-... The antimicrobial peptide LL-37 is the only cathelicidin that has been described in humans. LL-37 exerts chemotactic, immunomodulatory and angiogenic effects; activities that are mediated through binding to the formyl peptide receptor like (FPRL)-1 receptor. Agonistic ligation of FPRL-1 can also induce down-regulation of HIV-1 chemokine receptors and reduce susceptibility to HIV-1 infection in vitro. Therefore, we have evaluated the capacity of LL-37 to inhibit HIV-1 infection in vitro. Here we demonstrate that LL-37 inhibits HIV-1 replication in PBMC, including primary CD4(+) T cells. This inhibition was readily reproduced using various HIV-1 isolates without detectable changes in the target cell expression of HIV-1 chemokine receptors. Accordingly, the HIV-1 inhibitory effect was shown to be independent of FPRL-1 signalling. Given the epithelial expression of LL-37, it may contribute to the local protection against HIV-1 infection. read more read less

Topics:

Chemokine receptor (62%)62% related to the paper, Formyl peptide receptor (60%)60% related to the paper, Receptor (55%)55% related to the paper, Cathelicidin (55%)55% related to the paper, Chemotaxis (54%)54% related to the paper
186 Citations
Journal Article DOI: 10.2174/1570162033485122
Astrocyte infection by HIV-1: Mechanisms of restricted virus replication, and role in the pathogenesis of HIV-1-associated dementia
30 Sep 2003 - Current HIV Research

Abstract:

Astrocytes are the most numerous cell type in the brain, and their physiological roles are essential for normal brain function. Studies of post-mortem brain tissue samples from individuals with AIDS have revealed that a small proportion of astrocytes are infected by HIV-1 which is linked to the development of HIVassociated de... Astrocytes are the most numerous cell type in the brain, and their physiological roles are essential for normal brain function. Studies of post-mortem brain tissue samples from individuals with AIDS have revealed that a small proportion of astrocytes are infected by HIV-1 which is linked to the development of HIVassociated dementia (HIVD), a frequent clinical manifestation of HIV-1 disease affecting up to 20% of infected adults. However, astrocyte infection by HIV-1 in vivo is generally non-productive, and can only be readily detected by sensitive techniques that detect HIV-1 RNA or proviral DNA. Similarly, primary astrocyte cultures and astrocytic cell lines can be permissive to infection by HIV-1 strains, but are refractory to efficient HIV-1 expression. In efforts to delineate the molecular mechanisms underlying the restricted infection, several studies have demonstrated that efficient HIV-1 replication is blocked in astrocytes at different steps of the virus life cycle, including virus entry, reverse transcription, nucleocytoplasmic HIV-1 RNA transport, translation of viral RNA, and maturation of progeny virions. However, the relative importance of each of these possible replication blocks in restricting HIV-1 replication in astrocytes is unclear. Moreover, how restricted astrocyte infection contributes to the development of HIVD is unknown. This review surveys the current in vitro models of restricted HIV-1 replication in astrocytes, and provides an analysis of the available evidence supporting a role for astrocyte infection in the pathogenesis of HIVD. A greater understanding of the fate of HIV-1 in astrocytes may assist in the identification of viral reservoirs in the central nervous system, novel therapies for the treatment of HIVD, and also novel strategies to suppress HIV-1 replication in CD4+ cells of the immune system. read more read less

Topics:

Viral replication (56%)56% related to the paper, Viral entry (54%)54% related to the paper, Viral life cycle (52%)52% related to the paper, Astrocyte (52%)52% related to the paper, Reverse transcriptase (50%)50% related to the paper
178 Citations
Journal Article DOI: 10.2174/1570162052772924
HIV-1 TAR RNA: the target of molecular interactions between the virus and its host.
Sylvie Bannwarth1, Anne Gatignol
01 Jan 2005 - Current HIV Research

Abstract:

HIV-1 TAR RNA is the binding site of the viral protein Tat, the trans-activator of the HIV-1 LTR. It is present at the 5' end of all HIV-1 spliced and unspliced mRNAs in the nucleus as well as in the cytoplasm. It has a highly folded stem-bulge-loop structure, which also binds cellular proteins to form ribonucleoprotein compl... HIV-1 TAR RNA is the binding site of the viral protein Tat, the trans-activator of the HIV-1 LTR. It is present at the 5' end of all HIV-1 spliced and unspliced mRNAs in the nucleus as well as in the cytoplasm. It has a highly folded stem-bulge-loop structure, which also binds cellular proteins to form ribonucleoprotein complexes. The Tat-Cyclin T1-CDK9 complex is the main component in the trans-activation of HIV-1 and its affinity for TAR is regulated through Tat acetylation by histone acetyl transferases. Recent studies show that this complex is able to recruit other cellular partners to mediate efficient transcriptional elongation. TRBP, PKR and La bind directly to the TAR RNA structure and influence translation of HIV-1 in either positive or negative manners. Some mutations in TAR RNA severely impair HIV-1 trans-activation, translation and viral production, showing its functional importance. The overexpression or suppression of several TAR RNA- binding proteins has a strong impact on viral replication pointing out their major role in the viral life cycle. TAR RNA has been the target of drug development to inhibit viral replication. Recent data using small molecules or RNA-based technologies show that acting on the TAR RNA or on its viral and cellular binding factors effectively decreases virion production. read more read less

Topics:

RNA-dependent RNA polymerase (63%)63% related to the paper, RNA (62%)62% related to the paper, RNA silencing (62%)62% related to the paper, Non-coding RNA (61%)61% related to the paper, RNA-induced transcriptional silencing (61%)61% related to the paper
174 Citations
open accessOpen access Journal Article DOI: 10.2174/1570162X12666140129100411
PrEP Awareness and Perceived Barriers Among Single Young Men who have Sex with Men
José A. Bauermeister, Steven Meanley1, Emily S. Pingel1, Jorge H. Soler1, Gary W. Harper1
31 Mar 2014 - Current HIV Research

Abstract:

Pre-exposure prophylaxis (PrEP) has the potential to help reduce new HIV infections among young men who have sex with men (YMSM). Using a cross-sectional survey of YMSM (N=1,507; ages 18-24), we gauged YMSM's PrEP awareness and PrEP-related beliefs regarding side effects, accessibility, and affordability. Overall, 27% of the ... Pre-exposure prophylaxis (PrEP) has the potential to help reduce new HIV infections among young men who have sex with men (YMSM). Using a cross-sectional survey of YMSM (N=1,507; ages 18-24), we gauged YMSM's PrEP awareness and PrEP-related beliefs regarding side effects, accessibility, and affordability. Overall, 27% of the sample had heard about PrEP; 1% reported ever using PrEP prior to sex. In a multivariate logistic regression, we found that YMSM were more likely to have heard about PrEP if they were older, more educated, were residentially unstable in the prior 30 days, had insurance, or reported having at least one sexually transmitted infection in their lifetime. We found no differences by race/ethnicity, history of incarceration, or recent sexual risk behavior. In multivariate linear regression models, Black and Latino YMSM were more likely than Whites to state they would not use PrEP because of side effect concerns. YMSM were more likely to indicate that they would not be able to afford PrEP if they did not have insurance or if they had a prior sexually transmitted infection, PrEP rollout may be hindered due to lack of awareness, as well as perceived barriers regarding its use. We propose strategies to maximize equity in PrEP awareness and access if it is to be scaled up among YMSM. read more read less

Topics:

Men who have sex with men (54%)54% related to the paper
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174 Citations
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Current HIV Research format uses Vancouver citation style.

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Absolutely not! With our tool, you can freely write without having to focus on LaTeX. You can write your entire paper as per the Current HIV Research guidelines and autoformat it.

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One little Google search can get you the Word template for any journal. However, why do you need a Word template when you can write your entire manuscript on SciSpace, autoformat it as per Current HIV Research's guidelines and download the same in Word, PDF and LaTeX formats? Try us out!.

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To be honest, the answer is NO. The impact factor is one of the many elements that determine the quality of a journal. Few of those factors the review board, rejection rates, frequency of inclusion in indexes, Eigenfactor, etc. You must assess all the factors and then take the final call.

SHERPA/RoMEO Database

We have extracted this data from Sherpa Romeo to help our researchers understand the access level of this journal. The following table indicates the level of access a journal has as per Sherpa Romeo Archiving Policy.

RoMEO Colour Archiving policy
Green Can archive pre-print and post-print or publisher's version/PDF
Blue Can archive post-print (ie final draft post-refereeing) or publisher's version/PDF
Yellow Can archive pre-print (ie pre-refereeing)
White Archiving not formally supported
FYI:
  1. Pre-prints as being the version of the paper before peer review and
  2. Post-prints as being the version of the paper after peer-review, with revisions having been made.

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SciSpace would allow download of your references in Current HIV Research Endnote style, according to bentham-science guidelines.

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